Background: Lead exposure during early development causes neurodevelopmental
disorders by unknown mechanisms. Epidemiologic studies have focused recently on …

Prenatal exposure to lead (Pb) is known to decrease fetal growth; but its effects on postnatal
growth and mechanistic insights linking Pb to growth are not clearly defined. Genomically …

Lead (Pb) is a well-known toxicant that interferes with the development of a child's nervous
and metabolic systems and increases the risk of developing diseases later in life. Although …

Epigenetic processes, such as changes in DNA methylation, likely mediate the link between
environmental exposures in utero and altered gene expression. Differentially methylated …

The effects of prenatal lead exposure on child development include impaired growth and
cognitive function. DNA methylation might be involved in the underlying mechanisms and …

Abstract Background Cadmium (Cd) is a ubiquitous and environmentally persistent toxic
metal that has been implicated in neurotoxicity, carcinogenesis and obesity and essential …

Prenatal lead (Pb) exposure is associated with adverse developmental outcomes and to
epigenetic alterations such as DNA methylation and hydroxymethylation in animal models …

DNA methylation data assayed using pyrosequencing techniques are increasingly being
used in human cohort studies to investigate associations between epigenetic modifications …

Lead (Pb) exposure has adverse health effects and altered DNA methylation may contribute
to Pb toxicity. LINE-1 is an interspersed repeated DNA that is used as a surrogate marker for …

Background: Early-life exposure to lead is associated with deficits in neurodevelopment and
with hematopoietic system toxicity. DNA methylation may be one of the underlying …