JUURLINK, BHJ Response of glial cells to ischemia: roles of reactive oxygen species and
glutathione. NEUROSCI BIOBEHAV REV 21 (2) 151–166, 1997.—A review of reactive …

We have demonstrated that both astrocytes and cerebellar granule cell neurons die during
an ischemic insult only when there is complete loss of mitochondrial membrane potential …

HUANG, R., E. SOCHOCHKA AND L. HERTZ. Cell culture studies of the role of elevated
extracellular glutamate and K+ in neuronal cell death during and after anoxia/ischemia …

We prepared neuron-rich cultures from cortical and hippocampal CA1 regions of postnatal
day 1 (P1) rats. Using these cultures, we investigated the sensitivity of neurons to hypoxic …

J. Neurochem.(2011) 117, 654–664. Abstract Reactive oxygen species (ROS) are produced
early during apoptosis of cerebellar granule neurons induced by low potassium (K5) and …

Inflammatory‐activated glia are seen in numerous central nervous system (CNS)
pathologies and can kill nearby neurons through the release of cytotoxic mediators. Glia …

Neurons from cerebral cortex and hippocampal CA1 sector exhibit a striking difference in
vulnerability to transient ischemia. To establish whether this difference is due to the inherent …

Perhaps the most popular hypothesis regarding mechanisms which contribute to neuronal
death involves elevations in intracellular calcium ([CaZ'li). From a wide variety of …

Primary cortical neurones exposed to an oxidative insult in the form of hydrogen peroxide
(H2O2) for 30 min showed a concentration‐dependent increase in oxidative stress followed …

We have attempted to reconstruct in vitro the events that may occur in vivo during
reperfusion injury after ischemia in the central nervous system. The phenomenon is induced …