Neuroinflammation significantly contributes to brain injury and neurological deterioration
following intracerebral hemorrhage (ICH). MicroRNA-152 (miR-152) was reported to be …

Neuroinflammation plays a critical role in the pathogenesis of intracerebral hemorrhage
(ICH), contributing to detrimental brain injury and neurological function deficits. MicroRNA …

Oxidative stress and neuroinflammation are the key factors leading to secondary brain injury
after intracerebral hemorrhage (ICH). We investigated the effects of miR-27b, an oxidative …

NLRP3 inflammasome, the multimeric protein complexes involved in the processing of IL-1β
through Caspase-1 cleavage, facilitates the inflammatory response. The control and …

Recent studies suggest that microRNA-132 (miR-132) potentiates the cholinergic blockade
of inflammatory reactions by targeting acetylcholinesterase (AChE) and affords robust …

Background miR-126 is closely related to the occurrence of various complications after
intracerebral hemorrhage (ICH), but the molecular mechanism is not fully elucidated. This …

Sepsis is one of the most common causes of death in patients suffering from severe infection
or injury. Currently, a specific effective therapy remains to be established. In the present …

MiR-126, a microRNA implicated in blood vessel integrity, angiogenesis and vascular
inflammation, is markedly decreased in the sera of patients with intracerebral hemorrhage …

Autophagic activation mediated inflammation contributes to brain injury of intracerebral
hemorrhage (ICH). MiRNAs play a key role in inflammation, which negatively and …

Intracerebral hemorrhage (ICH) is a recognized central nervous system inflammation
complication. Several microRNAs (miRNAs or miRs) have been documented to be vital …