Objectives An enhanced sensitivity to angiotensin II in the renal circulation has been
demonstrated in the pre-hypertensive phase both in the spontaneously hypertensive rat and …

In a previous study we observed that the ability of intravenous infusions of prostaglandin I2
to attenuate vasoconstriction caused by intravenous infusions of angiotensin II was reduced …

The purpose of this study was to gain insight into the mechanism (s) responsible for the
exaggerated angiotensin II (ANG II)-induced renal vasoconstriction during the development …

Background Experimental evidence suggests that in conditions associated with an activated
renin–angiotensin system, unopposed activity of angiotensin II underlies the marked renal …

Mechanism of decreased vascular response to angiotensin II in renal vascular hypertension.
Compared to many forms of hypertension, vascular reactivity to angiotensin II (AII) is …

The effects of angiotensin II (Ang II) on renal hemodynamic function were first studied by
infusing exogenous Ang II [1]. Low doses of Ang II were shown to raise renal vascular …

The purpose of this study was to compare the effects of prostaglandin I2 on several
cardiovascular parameters and to compare the ability of prostaglandin I2 to modify …

The vascular responses to angiotensin II (Ang II) in the renal circulation are increased in
kidneys from rats with aortic coarctation compared with sham-operated rats. We have …

The objective of this study was to test the hypothesis that the vasodilator prostaglandins E2
(PGE2) and PGI2) participate in the mechanisms involved in the increased renal vascular …

Objective: To determine whether the effects of angiotensin I (AngI) in humans can be
explained entirely by its plasmatic conversion to angiotensin II (AngII). Methods: Ten healthy …