Background Nowadays, effects of fine particulate matter (PM 2.5) are well-documented and
related to oxidative stress and pro-inflammatory response. Nevertheless, epidemiological …

Our group was the first one reporting that autophagy could be triggered by airborne fine
particulate matter (PM) with a mean diameter of less than 2.5 μm (PM 2.5) in human lung …

Although positive associations exist between ambient particulate matter (PM 2.5; diameter≤
2.5 μm) and the morbidity and mortality rates for respiratory diseases, the biological …

A large number of studies have been conducted for clarifying toxicological mechanisms of
particulate matter (PM) aimed to investigate the physicochemical properties of PM and …

In order to clarify whether the mitochondrial dysfunction is closely related to the cell
homeostasis maintenance after particulate matter (PM 2.5) exposure, oxidative …

Nearly half of the world's population relies on combustion of solid biofuels to cover
fundamental energy demands. Epidemiologic data demonstrate that particularly long-term …

Exposure to higher levels of air pollution particulate matter (PM) with an aerodynamic
diameter of less than 2.5 μm (PM 2.5) links with an increased risk of cardiovascular and …

Epidemiological studies have associated the increase of respiratory and cardiovascular
mortality and morbidity with high levels of air pollution particulate matter (PM). However, the …

Background Fine particulate matter (PM2. 5) is a ubiquitous air pollutant, and it has been
reported to be closely associated with lung inflammatory injury. In this study, the potential …

Exposure to ambient fine particulate matter (< 2.5 μm; PM2. 5) increases the risk of the
physiopathology of vascular diseases. However, the underlying mechanism, particularly the …