The rat hippocampal slice preparation and its electrophysiology were used to assess the
toxicity of two sulfur-containing amino acids, l-cysteate (CA) and l-cysteine (CYS). Both …

Ischemia is believed to induce neuronal damage by causing a sustained increase in the
level of extracellular excitatory amino acids. In our study, we have examined the relationship …

We evaluated the effects of glycine exposure upon CA1-evoked response in the rat
hippocampal slice. Exposure to 10 mM glycine for 16 min produced rapid neuronal firing …

The role of the neutral amino acid glycine in excitotoxic neuronal injury is unclear. Glycine
coactivates glutamate N-methyl-D-aspartate (NMDA) receptors by binding to a distinct …

Abstract Effects of the sulphur‐containing acidic amino acids (SAAs) cysteic acid (CA),
homocysteic acid (HCA), cysteine sulphinic acid (CSA), homocysteine sulphinic acid …

Neuronal damage associated with cerebral ischemia and hypoglycemia might be the
consequence of the extracellular accumulation of excitatory amino acids. In previous studies …

Glucocorticoids (GCs) compromise the ability of hippocampal neurons to survive various
insults, and do so, at least in part, by exacerbating steps in the glutamate/N‐methyl‐D …

Baseline and stimulus-induced changes in [Ca2+] o and [K+] o as well as field potentials
(fp's) were studied during application of the excitatory amino acids kainate or glutamate, or …

The effects of glucose deficiency on (1) the K+-evoked release of glutamate and aspartate
and (2) excitatory synaptic transmission were studied in the Schaffer collateral-commissural …

Cysteine (CYS) is a non-essential amino acid which elicits excitotoxic properties via the N-
methyl-D-aspartate (NMDA) subtype of the glutamate receptor. CYS levels are known to be …