The p38 MAP kinases are stress-activated MAP kinases whose induction is often associated
with the onset of heart failure. This study investigated the role of p38 MAP kinase isoforms in …

Stress-responsive p38 MAP kinase is activated by phosphorylation during global and severe
regional myocardial ischemia. However, it is unknown whether or not moderate, low-flow …

The p38 mitogen-activated protein kinase (p38) is activated in the heart during ischemia–
reperfusion. However, it is not clear whether the activation of p38 is the protective response …

The current study focuses on the role of p38 MAP kinase in response to acute
preconditioning stimuli and ischemia. Exposure of the rat myoblast cell line H9C2 to …

Short duration exposure to cellular stresses have been shown to activate p38 mitogen-
activated protein kinase (MAPK) in cultured rat ventricular cardiomyocytes and isolated …

Stress-induced mitogen-activated protein kinase (MAP) p38 is activated in various forms of
heart failure, yet its effects on the intact heart remain to be established. Targeted activation of …

p38 mitogen-activated protein kinases (MAPKs) are serine/threonine specific protein
kinases that respond to cellular stress and regulate a broad range of cellular activities. There …

MYOCARDIAL ADAPTATION TO ISCHEMIA occurs through the activation of several tyrosine
kinases (3). Phosphorylation of tyrosine kinases has been shown to be linked with the …

p38 Mitogen-activated protein kinase (MAPK) is one of the most ancient signaling molecules
and is involved in multiple cellular processes, including cell proliferation, cell growth, and …

p38 mitogen-activated protein (MAP) kinase activities were significantly increased in mouse
hearts after chronic transverse aortic constriction, coincident with the onset of ventricular …