No-reflow phenomenon due to cardiac microvascular dysfunction or disturbance aggravates
clinic outcomes of a portion of patients with acute myocardial infarction undergoing …

Objective Proinflammatory cytokine TNF‐α during MI/R injury has been studied extensively.
However, how TNF‐α induces microvascular dysfunction in MI/R is still unclear. This study …

For patients with acute myocardial infarction (MI), the immediate therapeutic goal is to
establish the patency of the infarct-related artery. Prolonged myocardial ischemia, however …

Microthrombosis may be involved in the pathogenesis of cardiac microangiopathy due to
diabetes. Recent studies have shown that fibrinogen-like protein 2 (fgl2) plays a pivotal role …

Background Membrane-associated fibrinogen-like protein 2 (FGL2 prothrombinase, pFGL2)
is abundantly expressed in activated microvascular endothelial cells (MVECs) and plays a …

BACKGROUND: The no-reflow phenomenon is a deteriorating factor for prognosis of acute
myocardial infarction (AMI). Leukocyte enzymes may be involved in developing the no …

Introduction No reflow manifests coronary microvascular injury caused by continuous severe
myocardial ischemia and reperfusion. Microvascular obstruction (MVO) has emerged as one …

No-reflow or slow-flow post revascularization remains a challenge in interventional
cardiology. The “no-reflow” phenomenon is largely induced by microemboli of …

In patients with acute myocardial infarction (MI), coronary microvasculatures are often
damaged irreversibly due to myocardial ischemia and reperfusion, and flow to the previously …

Purpose To review (1) the mechanisms of coronary microvascular reperfusion injury,
particularly in the relationships between microvascular endothelium dysfunction …