The inhibition of cerebral monoamine oxidases (MAOs) by cigarette smoke components
could participate to the tobacco addiction. However, the actual extent of this inhibition in vivo …

We measured the concentration of brain monoamine oxidase B (MAO B; EC 1.4. 3.4) in 8
smokers and compared it with that in 8 non-smokers and in 4 former smokers using positron …

Although nicotine has been identified as the main ingredient in tobacco responsible for
aspects of the tobacco dependence syndrome, not all of the psychopharmacological effects …

Several studies have documented a strong association between smoking and depression.
Because cigarette smoke has been reported to inhibit monoamine oxidase (MAO) A in vitro …

Whole-body PET-scan studies in brains of tobacco smokers have shown a decrease in
monoamine oxidase (MAO) activity, which reverts to control level when they quit smoking …

Positron emission tomography (PET) studies with [11C] L-deprenyl-D2 have shown that
brain monoamine oxidase (MAO) B is 40% lower in smokers than in non-smokers. Here we …

Monoamine oxidase (MAO) is a mitochondrial outer-membrane flavoenzyme involved in
brain and peripheral oxidative catabolism of neurotransmitters and xenobiotic amines …

Background: There is a hypothesis that substances present in, or derived from, tobacco
smoke inhibit monoamine oxidase (MAO) in the brains of smokers, reducing the degradation …

OBJECTIVE: The authors' goal was to replicate a previous finding that smokers have lower
brain monoamine oxidase B (MAO-B) levels than comparison nonsmoking subjects and to …

The in vivo characteristics of [11C] befloxatone were assessed in myocardium of rats and
monkeys. A complete multicompartmental model was developed to quantify monkey cardiac …