Significant bodies of evidences have shown different mechanisms known to be the
etiological cause of Alzheimer's disease (AD) involving amyloid-beta protein accumulation …

The responses of central nervous system (CNS) cells such as neurons and glia in
neurodegenerative diseases (NDs) suggest that regulation of neuronal and glial functions …

The molecular mechanisms underlying neuronal dysfunction in Alzheimer's disease (AD)
remain uncharacterized. Here, we identify genes, molecular pathways and cellular …

Dementia is a neurodegenerative condition that is considered a major factor contributing to
cognitive decline that reduces independent function. Pathophysiological pathways are not …

One of the major obstacles confronting the formulation of a mechanistic understanding for
Alzheimer's disease (AD) is its immense complexity—a complexity that traverses the full …

Alzheimer's disease is characterized by sustained neuroinflammation leading to memory
loss and cognitive decline. The past decade has witnessed tremendous efforts in …

This review demonstrated that ischemic brain injury induces chronic changes in blood-brain
barrier in the gray and white matter. This insufficiency of blood-brain barrier may allow entry …

Alzheimer's disease (AD) is the most common neurodegenerative disease, affecting millions
of people worldwide. The classical hallmarks of AD include extracellular beta-amyloid (Aβ) …

Alzheimer's disease (AD) is a chronic neurodegenerative disorder characterized by the
pathological accumulation of amyloid beta (Aβ) peptides and neurofibrillary tangles …

An update of the etiology of Alzheimer's disease (AD): The current theory of the etiology of
AD and the guidelines for most of the wide-ranging treatments activities are built around …