Fetuin-A is an important inhibitor of extraosseous calcification, but some of the studies that
used ELISAs did not identify a significant relationship between serum fetuin-A levels and …

Reduced serum levels of the calcification inhibitor fetuin-A associate with increased
cardiovascular mortality in dialysis patients. Fetuin-A–deficient mice display calcification of …

Background: Cardiovascular disease (CVD) is the leading cause of mortality in hemodialysis
(HD). An elevated incidence of cardiovascular calcifications (CVC) is observed in HD. Fetuin …

Vascular calcification (VC) is an important reason for the high burden of vascular disease
among chronic dialysis patients. Chronic kidney disease (CKD) is associated with increased …

Vascular calcification predicts an increased risk for cardiovascular events/mortality in
atherosclerosis, diabetes, and ESRD. Serum concentrations of α 2-Heremens-Schmid …

In past decades, extraosseous (including cardiovascular) calcification was predominantly
regarded as a passive process of limited pathophysiological significance. It was thought that …

Fetuin-A and extraosseous calcification in uremia : Current Opinion in Nephrology and
Hypertension Fetuin-A and extraosseous calcification in uremia : Current Opinion in Nephrology …

Fetuin-A is a liver-derived plasma protein involved in calcified matrix metabolism. Fetuin-A
mediates the formation and stabilization of calciprotein particles (CPPs), soluble colloids …

OBJECTIVE Low levels of fetuin-A, a systemic calcification inhibitor, are linked to mortality in
patients on dialysis. In contrast, elevated fetuin-A is associated with cardiovascular events in …

Abstract Background. Fetuin-A (α2-Heremans Schmid glycoprotein) has recently been
identified as a circulating inhibitor of calcification and is regulated as a negative acute phase …