We tested the hypothesis that decreased tonic release of nitric oxide (NO) or a NO-
containing compound, during postischemic delayed hypoperfusion, would result in an …

We tested the hypothesis that inhibition of nitric oxide synthase activity in brain before
ischemia alters cerebral blood flow and decreases brain injury after 4 hours of middle …

We previously reported that L-arginine infusion increased pial vessel diameter by nitric
oxide-dependent mechanisms, improved regional cerebral blood flow (rCBF) distal to …

The purpose of this study was to investigate the relationship between cerebral blood flow
(CBF) and nitric oxide (NO) synthesis using a rat model of transient forebrain ischemia of …

The increase in cerebral blood flow (CBF) elicited by moderate hypoxia in anesthetized
animals is little attenuated by nitric oxide (NO) synthase inhibitors. However, in previous …

This study investigated whether the nitric oxide synthase inhibitor NG-nitro-L-arginine-
methyl ester (L-NAME) would alter blood flow and oxygen balance in the ischemic …

We tested the hypothesis that inhibition of nitric oxide synthase (NOS) activity in brain before
ischemia decreases postischemic hyperemia. Pentobarbital-anesthetized piglets underwent …

The effects of nitric oxide synthase inhibition on brain acidosis, regional cortical blood flow
(rCBF), and NADH redox state were examined using in vivo fluorescence imaging during …

We tested the hypothesis that NO synthase inhibition with N omega-nitro-L-arginine methyl
ester (L-NAME) and alpha 2-adrenoreceptor stimulation with dexmedetomidine (Dex) …

This study was undertaken to determine the extent to which nitric oxide (NO) mechanisms
are involved in cerebral hyperemia following global brain ischemia. The vertebral arteries …