Cellular entry of SARS-CoV-2 is thought to occur through the binding of viral spike S1
protein to ACE2. The entry process involves priming of the S protein by TMPRSS2 and …

With a high incidence of acute kidney injury among hospitalized COVID-19 patients,
considerable attention has been focussed on whether SARS-CoV-2 specifically targets …

SARS-CoV-2 entry into host cells is facilitated by the interaction between the receptor-
binding domain of its spike protein (CoV2-RBD) and host cell receptor, ACE2, promoting …

SARS-CoV-2 entry into host cells is mediated by the Spike (S) protein of the viral envelope.
The S protein is composed of two subunits: S1 that induces binding to the host cell via its …

Severe acute respiratory syndrome (SARS) coronavirus 2 (SARS-CoV2), which causes the
disease COVID-19, has caused an unprecedented global pandemic. Angiotensin-converting …

Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is the causative agent of
COVID-19, so understanding its biology and infection mechanisms is critical to facing this …

Thromboinflammation is the major cause of morbidity and mortality in COVID‐19 patients,
and post‐mortem examination demonstrates the presence of platelet‐rich thrombi and …

Purinergic receptors and NOD-like receptor protein 3 (NLRP3) inflammasome regulate
inflammation and viral infection, but their effects on severe acute respiratory syndrome …

Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) rapidly rampaged
worldwide, causing a pandemic of coronavirus disease (COVID-19), but the biology of SARS …

ACE2, the putative receptor for the novel coronavirus (2019-nCoV), played an important role
in cell entry of 2019-nCoV. However, it is not yet clear what cell types within the human body …