Chemical inhibition of neurons in the nucleus accumbens shell (AcbSh) elicits intense,
behaviorally specific, feeding in satiated rats. We have demonstrated previously that this …

Acquisition of drug-reinforced behavior is accompanied by a systematic increase of release
of the neurotransmitter acetylcholine (ACh) rather than dopamine, the expected prime …

Injection of the GABAA receptor agonist muscimol into the nucleus accumbens shell (AcbSh)
elicits robust feeding in satiated rats, but has no effect on water intake. The current study was …

Work over the past decade has supported the idea that discrete aspects of appetitive
motivation are differentially mediated by separate but interacting neurochemical systems …

To investigate the role of corticolimbic input in modulating feeding-related nucleus
accumbens (Acb) circuitry, researchers temporarily deactivated sites within the basolateral …

Nucleus accumbens μ-opioid stimulation leads to robust increases in the intake of highly
palatable foods, such as a high-fat diet. While interactions between opioids and certain …

The current studies were designed to evaluate whether incentive motivation for palatable
food is altered after manipulations of opioid, GABAergic, and dopaminergic transmission …

Although they comprise only a small portion of the neurons in the region, cholinergic
interneurons in the dorsal striatum appear to play an important role in the regulation of …

Previous work has demonstrated that injections of the γ-aminobutyric acid A (GABA A)
agonist muscimol into the nucleus accumbens shell (AcbSh) induce pronounced increases …

To determine if acetylcholine (ACh) is released in the nucleus accumbens in response to a
conditioned stimulus (CS) that reminds the animal of an aversive event, in vivo microdialysis …