Animal models of Parkinson's disease (PD) have shown that key mechanisms of cortical
plasticity such as long-term potentiation (LTP) and long-term depression (LTD) can be …

Alzheimer's disease (AD) is the most common type of dementia afflicting the elderly. In
addition to the presence of cortical senile plaques and neurofibrillary tangles, AD is …

Cerebrospinal fluid (CSF) concentrations of amyloid-ß (Aß), total tau (t-tau), and
phosphorylated tau proteins are associated with different clinical progression in Alzheimer's …

Secreted amyloid precursor protein-α (sAPPα) levels are reduced during the pathogenesis
of Alzheimer's disease, but the significance of this for neural function is not well understood …

The etiology of Alzheimer's disease (AD) remains elusive. The “amyloid” hypothesis states
that toxic action of accumulated β-amyloid peptide (Aβ) on synaptic function causes AD …

Hippocampal synaptic plasticity in the form of long‐term potentiation (LTP) and long‐term
depression (LTD) is likely to enable synaptic information storage in support of memory …

Amyloid beta (Aβ)-mediated synapse dysfunction is an early event in Alzheimer's disease
(AD) pathogenesis and previous studies suggest that NMDA receptor (NMDAR) …

Amyloid-β (Aβ)-induced changes in synaptic function in experimental models of Alzheimer's
disease (AD) suggest that Aβ generation and accumulation may affect fundamental …

Alzheimer's disease (AD) is a degenerative disorder that leads to progressive, irreversible
cognitive decline. It develops as a result of over-production and aggregation of β-amyloid …

ADNP syndrome, involving the ADNP transcription factor of the SWI/SNF chromatin-
remodeling complex, is characterized by developmental delay, intellectual disability, and …