Nucleus accumbens μ-opioid stimulation causes marked increases in the intake of highly
palatable foods, such as a high-fat diet. However, to date there has been little examination of …

Our previous studies have shown that stimulation of mu-opioid receptors within the nucleus
accumbens preferentially enhances intake of palatable food containing sucrose and fat; …

Infusion of a μ-opioid receptor (MOR) agonist into the nucleus accumbens (NAcc) drives
voracious food intake, an effect hypothesized to occur through increased tastant palatability …

The present study was designed to further investigate the nature of feeding induced by
opioid stimulation of the nucleus accumbens through an examination of the effects of intra …

Endogenous opioid peptides within the nucleus accumbens, a forebrain site critical for the
regulation of reward-related behavior, are believed to play an important role in the control of …

There is a long-standing interest in the role of endogenous opioid peptides in feeding
behavior and, in particular, in the modulation of food reward and palatability. Since drugs …

Rationale Previous work has shown that stimulation of GABAergic, opioid, or dopaminergic
systems within the nucleus accumbens modulates food intake and food-seeking behavior …

Microinjection of opioid agonists, such as morphine, into the nucleus accumbens shell
produces increases in eating behavior (ie 'wanting'for food). This study (1) reports direct …

Previous research has demonstrated that administration of μ-opioid receptor agonists into
the nucleus accumbens increases high-fat diet consumption in sated rats and has shown a …

Rationale Whereas reward-modulatory opioid actions have been intensively studied in
subcortical sites such as the nucleus accumbens (Acb), the role of cortical opioid …