Coexposures to asbestos and cigarette smoke cause increased risks of lung cancer in
asbestos workers. Although these carcinogens cause DNA damage to epithelial cells in vitro …

Exposure to airborne fine particles (PM2. 5) is implicated in excess of 50 000 yearly deaths
in the USA as well as a number of chronic respiratory illnesses. Despite intense interest in …

Polycyclic aromatic hydrocarbons (PAHs) associated with particulate matter (PM) may
induce oxidative damage via reactive oxygen species (ROS) generation. However, the …

Genotoxic stress from environmental pollutants plays a critical role in cytotoxicity. The most
abundant nitro-polycyclic aromatic hydrocarbon in environmental pollutants, 1-nitropyrene …

This book is based on the papers presented at the conference on" Mecha nisms of DNA
Damage and Repair: Implications for Carcinogenesis and Risk Assessment," held at the …

Numerous epidemiological studies have demonstrated a positive association between
ambient air pollution and adverse health effects including respiratory morbidity, asthma, and …

The impact of air pollution exposure on the level of total DNA adducts in human white blood
cells (WBCs) was evaluated in two populations in the Czech Republic and compared to the …

Epidemiological studies conducted in metropolitan areas have demonstrated that exposure
to environmental air pollution is associated with increases in mortality. Carcinogenic …

Ozone (O3) is a well-known oxidant pollutant present in photochemical smog. Although
ozone is suspected to be a respiratory carcinogen it is not regulated as a carcinogen in most …

Genotoxic effects of traffic-generated particulate matter (PM) are well described, whereas
little data are available on PM from combustion of biomass and wood, which contributes …