To determine whether acute left ventricular failure associated with myocardial infarction
leads to architectural changes in the spared nonischemic portion of the ventricular wall …

To determine whether acute left ventricular failure associated with myocardial infarction
leads to architectural changes in the spared nonischemic portion of the ventricular wall …

To determine whether the hypertrophic response of the surviving myocardium after infarction
leads to normalization of ventricular hemodynamics and wall stress, the left coronary artery …

To determine whether left ventricular failure after acute myocardial infarction is associated
with a growth response of the myocytes that tends to compensate for the loss of muscle …

schaemic heart disease is a complex and diverse clinical syndrome in which an imbalance
between blood supply I and demand is created by partial or complete occlusion of a major …

Three days after myocardial infarction involving 57% of the left ventricle in rats, the viable
tissue of the left ventricle expanded 29%, whereas myocardial hypertrophy in the right …

To determine the effects of acute myocardial infarction on the extent and distribution of
systolic and diastolic wall stress on the surviving myocardium, coronary artery occlusion was …

BACKGROUND Ischemic cardiomyopathy is characterized by myocyte loss, reactive cellular
hypertrophy, and ventricular scarring. However, the relative contribution of these tissue and …

This report describes early and later structural changes that occur in infarcted and
noninfarcted ventricular myocardium after coronary arterial ligation in the rat. Histologic …

To determine the effects of long-term pressure overload on the structural mechanisms
implicated in wall remodeling of the right ventricle, a mild pulmonary artery banding was …