Objective Intracerebral hemorrhage carries a high mortality and survivors are frequently left
with significant disability. Immunological mechanisms may play an important role in …

Objective The complement cascade is activated early following intracerebral hemorrhage
(ICH) and causes acute brain injury. We intended to explore the effects of plasma …

Objective: The complement cascade is activated and contributes to the brain injury after
intracerebral hemorrhage (ICH). Complement component 4 (C4), an important component of …

Background and Purpose: Spontaneous intracerebral hemorrhage (sICH) is a common form
of hemorrhagic stroke, with high mortality and morbidity. Pathophysiological mechanisms in …

Intracerebral hemorrhage (ICH) is a stroke subtype with high rates of mortality and morbidity.
The immune system, particularly complement and cytokine signaling, has been implicated in …

Background Inflammatory responses to intracerebral haemorrhage (ICH) are potential
therapeutic targets. We aimed to quantify molecular markers of inflammation in human brain …

Growing evidences suggest that stroke is a systemic disease affecting many organ systems
beyond the brain. Stroke-related systemic inflammatory response and immune …

Intracerebral hemorrhage (ICH) is a devastating type of stroke that lacks a specific treatment.
An intense immune response develops after ICH, which contributes to neuronal injury …

Background Symptomatic intracranial hemorrhage (sICH) is a terrible complication after
intravenous alteplase in stroke, and numerous biomarkers have been investigated …

Background Intracerebral hemorrhage (ICH) is a devastating disease that carries a 30 day
mortality of approximately 45%. Only 20% of survivors return to independent function at 6 …