Several microbial pathogens can modulate the host apoptotic response to infection, which
may contribute to immune evasion. Various studies have reported that infection with the …

The human pathogen Neisseria gonorrhoeae recruits and interacts extensively with
polymorphonuclear leukocytes (PMNs) during infection. N. gonorrhoeae is able to survive …

Several bacterial pathogens persist and survive in the host by modulating host cell death
pathways. We previously demonstrated that Neisseria gonorrhoeae, a Gram-negative …

The human‐adapted organism Neisseria gonorrhoeae is the causative agent of gonorrhoea,
a sexually transmitted infection. It readily colonizes the genital, rectal and nasalpharyngeal …

Following infection with Neisseria gonorrhoeae, bacteria may ascend into the Fallopian
tubes (FT) and induce salpingitis, a major cause of infertility. In the FT, interactions between …

Neisseria gonorrhoeae is a highly adapted human pathogen that utilises multiple adhesins
to interact with a variety of host cell receptors. Recently, substantial progress has been made …

Neisseria gonorrhoeae (gonococcus) infection results in recruitment of polymorphonuclear
leukocytes (PMNs) to the urethral lumen. Recent work from our laboratory demonstrated that …

Infection of human urethral epithelial cells (UECs) with Neisseria gonorrhoeae increases the
transcription of several host antiapoptotic genes, including bfl-1, cox-2, and c-IAP-2. In order …

Neisseria gonorrhoeae, the human obligate pathogen responsible for the sexually
transmitted disease gonorrhea, has evolved several mechanisms to evade the host immune …

In order to better understand the host response to an infection with Neisseria gonorrhoeae,
microarray technology was used to analyse the gene expression profile between uninfected …