Reactive nitrogen species are thought to be involved in both hypoxic‐ischemic and cytokine‐
induced brain injury, including periventricular leukomalacia (PVL), the major pathological …

In the immature human brain, periventricular leukomalacia (PVL) is the predominant white
matter injury underlying the development of cerebral palsy. PVL has its peak incidence …

Periventricular leukomalacia (PVL), the major substrate of cerebral palsy in survivors of
prematurity, is defined as focal periventricular necrosis and diffuse gliosis in immature …

Periventricular leukomalacia is the predominant injury in the preterm infant leading to
cerebral palsy. Oxygen exposure may be an additional cause of brain injury in these infants …

Periventricular leukomalacia (PVL) is a lesion of the immature cerebral white matter in the
perinatal period and associated predominantly with prematurity and cerebral …

Periventricular leukomalacia, the predominant pathological lesion underlying cerebral palsy
in premature infants, is thought to be the result of hypoxic–ischemic injury to the cerebral …

Periventricular leukomalacia (PVL) is the major substrate of cerebral palsy in survivors of
prematurity. Its pathogenesis is complex and likely involves ischemia/reperfusion in the …

Premature human infants frequently suffer from periventricular leukomalacia (PVL)
characterized by the loss of central myelinated tracts in the brain [Neuropathology, 22 (2002) …

Products of inflammation and the activation of nitric oxide synthase have been proposed as
a mechanism of oligodendrocyte injury in CNS inflammation. There are currently three well …

Hypoxia-ischemia (HI) is a leading cause of white matter damage, a major contributor to
cerebral palsy in premature infants. Preferential white matter damage is believed to result …