Lymphoid chemokines CCL19 and CCL21 are expressed in the central nervous system during experimental autoimmune encephalomyelitis: implications for the …
S Columba‐Cabezas, BS Elena, AF Aloisi - Brain pathology, 2003 - Wiley Online Library
The simultaneous presence of dendritic, T‐and B‐cells in the central nervous system (CNS)
of mice with experimental autoimmune encephalomyelitis (EAE), a model for multiple …
of mice with experimental autoimmune encephalomyelitis (EAE), a model for multiple …
CCR2+Ly-6Chi monocytes are crucial for the effector phase of autoimmunity in the central nervous system
A Mildner, M Mack, H Schmidt, W Brück, M Djukic… - Brain, 2009 - academic.oup.com
The chemokine receptor CCR2 plays a vital role for the induction of autoimmunity in the
central nervous system. However, it remains unclear how the pathogenic response is …
central nervous system. However, it remains unclear how the pathogenic response is …
Expression of CCR7 in multiple sclerosis: implications for CNS immunity
P Kivisäkk, DJ Mahad, MK Callahan… - Annals of Neurology …, 2004 - Wiley Online Library
It is unclear how immune cells traffic between the lymphoid compartment and the central
nervous system (CNS), which lacks lymphatic vessels and is shielded by the blood–brain …
nervous system (CNS), which lacks lymphatic vessels and is shielded by the blood–brain …
Effector T-cell trafficking between the leptomeninges and the cerebrospinal fluid
C Schläger, H Körner, M Krueger, S Vidoli, M Haberl… - Nature, 2016 - nature.com
In multiple sclerosis, brain-reactive T cells invade the central nervous system (CNS) and
induce a self-destructive inflammatory process. T-cell infiltrates are not only found within the …
induce a self-destructive inflammatory process. T-cell infiltrates are not only found within the …
Licensed in the Lungs
RM Ransohoff - Nature, 2012 - nature.com
Licensed in the lungs | Nature Skip to main content Thank you for visiting nature.com. You are
using a browser version with limited support for CSS. To obtain the best experience, we …
using a browser version with limited support for CSS. To obtain the best experience, we …
CNS lymphatic drainage and neuroinflammation are regulated by meningeal lymphatic vasculature
Neuroinflammatory diseases, such as multiple sclerosis, are characterized by invasion of the
brain by autoreactive T cells. The mechanism for how T cells acquire their encephalitogenic …
brain by autoreactive T cells. The mechanism for how T cells acquire their encephalitogenic …
Immune cells may fend off Alzheimer disease
M Britschgi, T Wyss-Coray - Nature medicine, 2007 - nature.com
Immune cells may fend off Alzheimer disease | Nature Medicine Skip to main content Thank you
for visiting nature.com. You are using a browser version with limited support for CSS. To obtain …
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IL-1R signaling within the central nervous system regulates CXCL12 expression at the blood-brain barrier and disease severity during experimental autoimmune …
Multiple sclerosis (MS) is an autoimmune disease of the CNS characterized by disruption of
the blood-brain barrier (BBB). This breach in CNS immune privilege allows undeterred …
the blood-brain barrier (BBB). This breach in CNS immune privilege allows undeterred …
The choroid plexus acts as an immune cell reservoir and brain entry site in experimental autoimmune encephalomyelitis
I Lazarevic, S Soldati, JA Mapunda, H Rudolph… - Fluids and Barriers of …, 2023 - Springer
The choroid plexus (ChP) has been suggested as an alternative central nervous system
(CNS) entry site for CCR6+ Th17 cells during the initiation of experimental autoimmune …
(CNS) entry site for CCR6+ Th17 cells during the initiation of experimental autoimmune …
Cxcl10+ monocytes define a pathogenic subset in the central nervous system during autoimmune neuroinflammation
Multiple sclerosis (MS) is characterized by pathological inflammation that results from the
recruitment of lymphoid and myeloid immune cells from the blood into the brain. Due to …
recruitment of lymphoid and myeloid immune cells from the blood into the brain. Due to …