Nitric oxide (NO) synthesized by the endothelial isoform of nitric oxide synthase (eNOS) is a
critical determinant of vascular homeostasis. However, the real-time detection of intracellular …

Endothelial nitric oxide synthase (eNOS) serves as a critical enzyme in maintaining vascular
pressure by producing nitric oxide (NO); hence, it has a crucial role in the regulation of …

Nitric oxide (NO) derived from endothelial NO synthase (eNOS) mediates vascular
endothelial growth factor (VEGF)‐stimulated endothelial cytoskeleton remodeling and …

Endothelial nitric-oxide synthase (eNOS) undergoes a complex pattern of post-translational
modifications that regulate its activity. We have recently reported that eNOS is constitutively …

Endothelial NOS (eNOS), and its product NO, are vital components of the control of
vasomotor function and cardiovascular homeostasis. In the present review, we will take a …

Endothelial NO synthase (eNOS) is critically modulated by kinases via the phosphorylation
of its Ser 1179 (bovine) or Ser 1177 (human) residue. Reactive oxygen species such as H 2 …

The endothelial isoform of nitric-oxide synthase (eNOS) undergoes a complex pattern of
covalent modifications, including acylation with the fatty acids myristate and palmitate as …

Nitric oxide is a versatile molecule, with its actions ranging from haemodynamic regulation to
anti‐proliferative effects on vascular smooth muscle cells. Nitric oxide is produced by the …

Background: Current paradigms suggest that nitric oxide (NO) produced by endothelial cells
(ECs) through endothelial nitric oxide synthase (eNOS) in the vessel wall is the primary …

Endothelium-derived nitric oxide (NO) is primarily attributable to constitutive expression of
the endothelial nitric oxide synthase (eNOS) gene. Although a more comprehensive …