This chapter summarises advances made over the last 12 years regarding our
understanding of the pathophysiology and its clinical implications in acute mountain …

The diagnosis, treatment and prevention of high altitude cerebral edema (HACE) are fairly
well established. The major unresolved issues are 1) the pathophysiology, 2) the individual …

Anyone who travels to high altitude, whether a recreational hiker, skier, mountain climber,
military personnel, or rescue worker, is at risk of developing high-altitude illness. Acute …

Hypoxia affects all organ systems, but primarily the brain. On ascent to high altitude,
hypoxemia triggers time-dependent processes causing acute mountain sickness (AMS), a …

After its publication, a number of international recognized doctors and scientists in the field
have raised serious concerns in regards to the accuracy or misleading nature of the …

This review discusses the impact of recent publications on pathophysiologic concepts and
on practical aspects of acute mountain sickness (AMS). Magnetic resonance imaging …

High-altitude pulmonary edema (HAPE), along with cerebral edema (CE), constitutes one of
the major complications of acute mountain sickness (AMS). Although the syndrome was first …

After ascent to high altitude (≥ 2500 m), the inability of the human body to adapt to the
hypobaric and hypoxia environment can induce tissue hypoxia, then a series of high altitude …

Acute mountain sickness (AMS) and high-altitude cerebral edema (HACE) are essentially
neurological disorders. The five symptoms included in the Lake Louise AMS symptom score …

It is now generally accepted that acute mountain sickness (AMS) is caused by a mild form of
high altitude cerebral edema (HACE). AMS may progress to a potentially lethal form of …