Aim: Prenatal alcohol exposure (PAE) can cause fetal alcohol spectrum disorders (FASD).
Previously, we assessed PAE in brain tissue from mouse models, however whether these …

Background Prenatal alcohol exposure is the leading preventable cause of behavioral and
cognitive deficits, which may affect between 2 and 5% of children in North America. While …

Background Fetal alcohol spectrum disorder (FASD) is a developmental disorder that
manifests through a range of cognitive, adaptive, physiological, and neurobiological deficits …

Abstract Rodent models of Fetal Alcohol Spectrum Disorders (FASD) have revealed that
prenatal alcohol exposure (PAE) results in differential DNA cytosine methylation in the …

Background: Prenatal alcohol exposure (PAE) can alter the development of neurobiological
systems, leading to lasting neuroendocrine, neuroimmune, and neurobehavioral deficits …

Background: Binge-level prenatal alcohol exposure (PAE) causes developmental
abnormalities, which may be mediated in part by epigenetic mechanisms. Despite this, few …

Alcohol exposure during development can cause variable neurofacial deficit and growth
retardation known as fetal alcohol spectrum disorders (FASD). The mechanism underlying …

Aim: Fetal alcohol spectrum disorder (FASD) involves prenatal growth delay, impaired facial
and CNS development and causes severe clinical, social-economic burdens. Here, we aim …

Background Based upon experimental animal studies, the neurodevelopmental
abnormalities associated with prenatal alcohol exposure (PNAE)/fetal alcohol spectrum …

BACKGROUND: Epigenetic changes are believed to be among the earliest key regulators
for cell fate and embryonic development. To support this premise, it is important to …