The disposal of an oral glucose load in patients with non-insulin-dependent diabetes
E Ferrannini, DC Simonson, LD Katz, G Reichard Jr… - Metabolism, 1988 - Elsevier
Following glucose ingestion, tissue glucose uptake is enhanced and endogenous glucose
production is inhibited, thus contributing to the maintenance of normal glucose tolerance. To …
production is inhibited, thus contributing to the maintenance of normal glucose tolerance. To …
The disposal of an oral glucose load in patients with non-insulin-dependent diabetes
E Ferrannini, DC Simonson, LD Katz… - Metabolism …, 1988 - metabolismjournal.com
Following glucose ingestion, tissue glucose uptake is enhanced and endogenous glucose
production is inhibited, thus contributing to the maintenance of normal glucose tolerance. To …
production is inhibited, thus contributing to the maintenance of normal glucose tolerance. To …
The disposal of an oral glucose load in patients with non-insulin-dependent diabetes.
E Ferrannini, DC Simonson, LD Katz, GJ Reichard… - 1988 - cabidigitallibrary.org
Glucose kinetics were studied after glucose was given by mouth to 12 diabetics not
dependent on insulin and 10 age-and weight-matched controls. The endogenous glucose …
dependent on insulin and 10 age-and weight-matched controls. The endogenous glucose …
The disposal of an oral glucose load in patients with non-insulin-dependent diabetes
E Ferrannini, DC Simonson, LD Katz… - Metabolism …, 1988 - pubmed.ncbi.nlm.nih.gov
Following glucose ingestion, tissue glucose uptake is enhanced and endogenous glucose
production is inhibited, thus contributing to the maintenance of normal glucose tolerance. To …
production is inhibited, thus contributing to the maintenance of normal glucose tolerance. To …
[引用][C] The disposal of an oral glucose load in patients with non-insulin-dependent diabetes
E FERRANNINI - Metabolism, 1988 - cir.nii.ac.jp
The disposal of an oral glucose load in patients with non-insulin-dependent diabetes.
E Ferrannini, DC Simonson, LD Katz… - Metabolism: Clinical …, 1988 - europepmc.org
Following glucose ingestion, tissue glucose uptake is enhanced and endogenous glucose
production is inhibited, thus contributing to the maintenance of normal glucose tolerance. To …
production is inhibited, thus contributing to the maintenance of normal glucose tolerance. To …
[引用][C] The disposal of an oral glucose load in patients with non-insulin-dependent diabetes
E FERRANNINI, DC SIMONSON… - Metabolism, clinical …, 1988 - pascal-francis.inist.fr
The disposal of an oral glucose load in patients with non-insulin-dependent diabetes CNRS
Inist Pascal-Francis CNRS Pascal and Francis Bibliographic Databases Simple search …
Inist Pascal-Francis CNRS Pascal and Francis Bibliographic Databases Simple search …
[引用][C] THE DISPOSAL OF AN ORAL GLUCOSE-LOAD IN PATIENTS WITH NON-INSULIN-DEPENDENT DIABETES
E Ferrannini, D Simonson, L Katz, G Reichard… - METABOLISM …, 1988 - arpi.unipi.it
THE DISPOSAL OF AN ORAL GLUCOSE-LOAD IN PATIENTS WITH NON-INSULIN-DEPENDENT
DIABETES IRIS IRIS Home Sfoglia Macrotipologie & tipologie Autore Titolo Riviste Serie IT …
DIABETES IRIS IRIS Home Sfoglia Macrotipologie & tipologie Autore Titolo Riviste Serie IT …
The disposal of an oral glucose load in patients with non-insulin-dependent diabetes
E Ferrannini, DC Simonson, LD Katz, G Reichard… - …, 1988 - scholars.uthscsa.edu
Following glucose ingestion, tissue glucose uptake is enhanced and endogenous glucose
production is inhibited, thus contributing to the maintenance of normal glucose tolerance. To …
production is inhibited, thus contributing to the maintenance of normal glucose tolerance. To …
[引用][C] The disposal of an oral glucose load in patients with non-insulin-dependent diabetes
E FERRANNINI - Metabolism, 1988 - cir.nii.ac.jp