Involvement of trigeminal ganglionic Nav1.7 in hyperalgesia of inflamed temporomandibular joint is dependent on ERK1/2 phosphorylation of glial cells in rats

RY Bi, XX Kou, Z Meng, XD Wang… - European Journal of …, 2013 - Wiley Online Library
Background Inflammation is a major cause of temporomandibular disorder‐related pain. The
N av1. 7 sodium channel has a critical function in pain perceptions. However, whether and …
被引用次数：24 相关文章

Involvement of trigeminal ganglionic Nav 1.7 in hyperalgesia of inflamed temporomandibular joint is dependent on ERK1/2 phosphorylation of glial cells in rats.

RY Bi, XX Kou, Z Meng, XD Wang, Y Ding… - European Journal of …, 2012 - europepmc.org
Background Inflammation is a major cause of temporomandibular disorder-related pain. The
Nav 1.7 sodium channel has a critical function in pain perceptions. However, whether and …

Involvement of trigeminal ganglionic Nav 1.7 in hyperalgesia of inflamed temporomandibular joint is dependent on ERK1/2 phosphorylation of glial cells in rats

RY Bi, XX Kou, Z Meng, XD Wang… - European journal of …, 2013 - pubmed.ncbi.nlm.nih.gov
Background Inflammation is a major cause of temporomandibular disorder-related pain. The
Nav 1.7 sodium channel has a critical function in pain perceptions. However, whether and …

Involvement of trigeminal ganglionic Nav1.7 in hyperalgesia of inflamed temporomandibular joint is dependent on ERK1/2 phosphorylation of glial cells in rats

RY Bi, XX Kou, Z Meng, XD Wang, Y Ding… - European Journal of …, 2013 - infona.pl
Background Inflammation is a major cause of temporomandibular disorder‐related pain. The
N av 1.7 sodium channel has a critical function in pain perceptions. However, whether and …