DNA Damage rather than type I ifn signaling is the primary mediator of neural dysfunction in aicardi-goutieres syndrome after Rnaseh2 disruption
Mutations in genes that function in nucleic metabolism have been shown to be linked to
Aicardi-Goutières syndrome. In this issue of Neuron, Aditi et al.(2021) provide evidence that
DNA damage-dependent signaling rather than type I interferon signaling underlies
neurodegeneration in this class of neurodevelopmental/neuroinflammatory disease.
Aicardi-Goutières syndrome. In this issue of Neuron, Aditi et al.(2021) provide evidence that
DNA damage-dependent signaling rather than type I interferon signaling underlies
neurodegeneration in this class of neurodevelopmental/neuroinflammatory disease.
Mutations in genes that function in nucleic metabolism have been shown to be linked to Aicardi-Goutières syndrome. In this issue of Neuron, Aditi et al. (2021) provide evidence that DNA damage-dependent signaling rather than type I interferon signaling underlies neurodegeneration in this class of neurodevelopmental/neuroinflammatory disease.
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