[引用][C] Differential activation of MAPK/ERK and p38/SAPK in neurons and glia following focal cerebral ischemia in the rat
I EA - Molecular Brain Research, 2000 - cir.nii.ac.jp
Differential activation of MAPK/ERK and p38/SAPK in neurons and glia following focal
cerebral ischemia in the rat | CiNii Research … Differential activation of MAPK/ERK and p38/SAPK
in neurons and glia following focal cerebral ischemia in the rat … Molecular Brain Research …
cerebral ischemia in the rat | CiNii Research … Differential activation of MAPK/ERK and p38/SAPK
in neurons and glia following focal cerebral ischemia in the rat … Molecular Brain Research …
Differential activation of MAPK/ERK and p38/SAPK in neurones and glia following focal cerebral ischaemia in the rat
EA Irving, FC Barone, AD Reith, SJ Hadingham… - Molecular Brain …, 2000 - Elsevier
Two relatively well characterised kinase signalling pathways are those involving MAPK/ERK
and p38/SAPK2, that are known to be activated in vitro by various factors known to increase
following stroke, such as glutamate, IL-1 and TNF. The present study was designed to
investigate the activation and cellular distribution of phosphorylated-ERK1/2,-p38 and the
transcription factor CREB following focal cerebral ischaemia using phosphospecific
antibodies. Up to 24 h following transient MCAO (90 min) and 6 h following permanent …
and p38/SAPK2, that are known to be activated in vitro by various factors known to increase
following stroke, such as glutamate, IL-1 and TNF. The present study was designed to
investigate the activation and cellular distribution of phosphorylated-ERK1/2,-p38 and the
transcription factor CREB following focal cerebral ischaemia using phosphospecific
antibodies. Up to 24 h following transient MCAO (90 min) and 6 h following permanent …
以上显示的是最相近的搜索结果。 查看全部搜索结果