Temporomandibular joint inflammation decreases the voltage-gated K+ channel subtype 1.4-immunoreactivity of trigeminal ganglion neurons in rats
M Takeda, T Tanimoto, M Nasu, S Matsumoto - European journal of pain, 2008 - Elsevier
Voltage-gated K+ (Kv) channels are one of the important physiological regulators of the
membrane potentials in excitable cells, including sensory ganglion neurons. The aim of the
present study was to investigate whether temporomandibular joint (TMJ) inflammation alters
expression of Kv channel subtype 1.4 (Kv1. 4) of trigeminal ganglion (TRG) neurons
innervating TMJ relating allodynia (pain caused by normally innoxious stimulation), by using
both behavioral and immunohistochemical techniques. TMJ inflammation was induced by …
membrane potentials in excitable cells, including sensory ganglion neurons. The aim of the
present study was to investigate whether temporomandibular joint (TMJ) inflammation alters
expression of Kv channel subtype 1.4 (Kv1. 4) of trigeminal ganglion (TRG) neurons
innervating TMJ relating allodynia (pain caused by normally innoxious stimulation), by using
both behavioral and immunohistochemical techniques. TMJ inflammation was induced by …