Myocardial infarction (MI) poses a significant risk for sudden cardiac death. The
effectiveness of angiotensin‐converting enzyme (ACE) inhibitors and AT1 receptor blockade …

Objectives: In an ovine model of left ventricular (LV) remodeling after transmural
anteroapical myocardial infarction (MI), we have previously demonstrated that the …

To characterize the temporal activation of the renin-angiotensin system after myocardial
infarction (MI) in rabbits, we examined cardiac ANG II type 1 receptor (AT 1 R) expression …

The relative contribution of the angiotensin II type 1 and 2 receptors (AT1-R and AT2-R) in
postmyocardial infarction (MI) remodeling remains incompletely understood. We studied five …

Left ventricular hypertrophy (LVH) is considered to be an independent risk factor giving rise
to ischemia, arrhythmia, and left ventricular dysfunction. In this article, we summarize recent …

The efficacy of angiotensin converting enzyme (ACE) inhibitors is well known to prevent the
formation of angiotensin II (Ang II) by these agents. The objective of the present study was to …

Background Myocardial ischemia is known to upregulate the systemic renin-angiotensin
system, which influences myocardial ischemic events by affecting hemodynamics and …

There are two subtypes of angiotensin (Ang) II receptors, AT1R and AT2R. It is established
that clinical use of specific AT1R blocker (ARB) improves the long-term prognosis of heart …

The angiotensin AT2-receptor mediates tissue protective actions. Its regenerative potential
has been tested in multiple disease models including models of myocardial infarction. These …

Background—Angiotensin II (Ang II) has been implicated in ventricular remodeling after
myocardial infarction (MI), which is an important determinant for prognosis after MI. The aim …