[引用][C] Influence of age or circadian time on bcl-2 and bax mRNA expression in the rat hippocampus after corticosterone exposure.

増井晃 - Brain Research Bulletin (in press) - cir.nii.ac.jp
Influence of age or circadian time on bcl-2 and bax mRNA expression in the rat
hippocampus after corticosterone exposure. | CiNii Research CiNii 国立情報学研究所 学術 …

Influence of age or circadian time on bcl-2 and bax mRNA expression in the rat hippocampus after corticosterone exposure

A Yoshimura, A Masui, S Jinde, H Kanai, N Kato… - Brain research …, 2007 - Elsevier
A rapid elevation in the level of endogenous corticosterone (CORT) functions in the stress
response associated with the hypothalamus–pituitary–adrenal axis, and it has been well …

Corticosterone differentially regulates bax, bcl-2 and bcl-x mRNA levels in the rat hippocampus

SP Cardenas, C Parra, J Bravo, P Morales, HE Lara… - Neuroscience …, 2002 - Elsevier
It has previously been shown that adrenalectomy (ADX) produces apoptosis in the granule
cell of the dentate gyrus (DG), and that this effect is prevented by corticosterone …

Hippocampal Bcl-2 expression is selectively increased following chronic but not acute treatment with antidepressants, 5-HT1A or 5-HT2C/2B receptor antagonists

F Murray, PH Hutson - European journal of pharmacology, 2007 - Elsevier
Expression of the anti-apoptotic protein Bcl-2 has been shown to increase in the
hippocampus and cortex following chronic administration of mood stabilisers such as lithium …

Free radical‐dependent nuclear localization of Bcl‐2 in the central nervous system of aged rats is not associated with Bcl‐2‐mediated protection from apoptosis

JA Kaufmann, M Perez, WR Zhang… - Journal of …, 2003 - Wiley Online Library
We have previously reported that Bcl‐2 is up‐regulated in the CNS of aged F344 rats as a
consequence of oxidative stress. In addition to increased levels of expression, we now …

Repeated unpredictable stress and antidepressants differentially regulate expression of the bcl-2 family of apoptotic genes in rat cortical, hippocampal, and limbic …

TA Kosten, MP Galloway, RS Duman… - …, 2008 - nature.com
Apoptosis has been proposed as a contributing cellular mechanism to the structural
alterations that have been observed in stress-related mood disorders. Antidepressants, on …

Differential expression of rat brain bcl-2 family proteins in development and aging

S Shimohama, S Fujimoto, Y Sumida… - … and biophysical research …, 1998 - Elsevier
We have previously examined the involvement of the B cell leukemia-2 gene product (Bcl-2)
family proteins (Bcl-2, Bcl-x, Bax, Bak, and Bad) in Alzheimer's disease (AD) and found that …

Chronic corticosterone manipulations in mice affect brain cell proliferation rates, but only partly affect BDNF protein levels

J Prickaerts, DLA van den Hove, FLP Fierens, HK Kia… - Neuroscience …, 2006 - Elsevier
We investigated whether the effects of corticosterone (CORT) on brain cell proliferation are
mediated via its detrimental effect on brain-derived neurotrophic factor (BDNF). Using a [3H] …

Increase of bcl-2 protein in neuronal dendritic processes of cerebral cortex and hippocampus by the antiparkinsonian drugs, talipexole and pramipexole

K Takata, Y Kitamura, J Kakimura, Y Kohno… - Brain research, 2000 - Elsevier
Treatment of rats for 4 days with the antiparkinsonian drugs, talipexole and pramipexole,
markedly increased Bcl-2 immunoreactivity in neuronal dendritic processes in both cerebral …

Bcl-2, Bax, and Bcl-x expression in the CA1 area of the hippocampus following transient forebrain ischemia in the adult gerbil

I Ferrer, E Lopez, R Blanco, R Rivera… - Experimental brain …, 1998 - Springer
Delayed neuronal death was produced in the CA1 area of the hippocampus following 5 min
of forebrain ischemia in adult gerbils. Immunohistochemistry and Western blotting to Bcl-2 …