Background Secondary damage after spinal cord injury (SCI) is characterized by a cascade
of events including hemorrhage, apoptosis, oxidative stress, and inflammation which …

Background Spinal cord injury (SCI) is a severe traumatic injury that often leads to paralysis.
The neuroinflammation following SCI plays an important role during the secondary injury …

Inflammation plays a critical role in the secondary degenerative response to spinal cord
injury (SCI). The influx of inflammatory cells following SCI is preceded by the expression of …

Spinal cord injury (SCI) causes loss of normal sensation and often leads to debilitating
neuropathic pain (NeP). Chronic NeP develops at or below the SCI lesion in as many as …

Background Spinal cord injury (SCI) is a severe traumatic disease which causes high
disability and mortality rates. The molecular pathological features after spinal cord injury …

Injury to the spinal cord is followed by degeneration, which leads to progressive tissue loss
and usually cystic cavitation. Cellular and humoral immune responses have been implicated …

The inflammatory response initiated after spinal cord injury (SCI) is characterized by the
accumulation of macrophages at the impact site. Monocyte chemoattractant protein‐1 (MCP …

Spinal cord injury (SCI) results in acute inflammatory responses and secondary damages,
including neuronal and glial cell death, axonal damage and demyelination, and blood-brain …

Background Peripheral nerve injuries stimulate the regenerative capacity of injured neurons
through a neuroimmune phenomenon termed the conditioning lesion (CL) response. This …

Background Acute spinal cord injury (SCI) could cause mainly two types of pathological
sequelae, the primary mechanical injury, and the secondary injury. The macrophage in SCI …