Pathophysiology of sickle cell disease: role of cellular and genetic modifiers

MH Steinberg, GP Rodgers - Seminars in hematology, 2001 - Elsevier
Sickle hemoglobin (HbS), caused by a point mutation in the β-globin gene of hemoglobin,
polymerizes when deoxygenated. The pathophysiology of sickle cell disease results from …
被引用次数：176 相关文章

Pathophysiology of sickle cell disease: role of cellular and genetic modifiers

MH Steinberg, GP Rodgers - Seminars in hematology, 2001 - pubmed.ncbi.nlm.nih.gov
Sickle hemoglobin (HbS), caused by a point mutation in the beta-globin gene of
hemoglobin, polymerizes when deoxygenated. The pathophysiology of sickle cell disease …

[引用][C] Pathophysiology of sickle cell disease: Role of cellular and genetic modifiers

MH STEINBERG, GP RODGERS - Seminars in hematology, 2001 - pascal-francis.inist.fr
Pathophysiology of sickle cell disease : Role of cellular and genetic modifiers CNRS Inist
Pascal-Francis CNRS Pascal and Francis Bibliographic Databases Simple search Advanced …

Pathophysiology of sickle cell disease: role of cellular and genetic modifiers.

MH Steinberg, GP Rodgers - Seminars in Hematology, 2001 - europepmc.org
Sickle hemoglobin (HbS), caused by a point mutation in the beta-globin gene of
hemoglobin, polymerizes when deoxygenated. The pathophysiology of sickle cell disease …

Pathophysiology of sickle cell disease: Role of cellular and genetic modifiers

MH Steinberg, GP Rodgers - Seminars in Hematology, 2001 - infona.pl
Sickle hemoglobin (HbS), caused by a point mutation in the β-globin gene of hemoglobin,
polymerizes when deoxygenated. The pathophysiology of sickle cell disease results from …

[引用][C] Pathophysiology of sickle cell disease: Role of cellular and genetic modifiers

MH STEINBERG, GP RODGERS - Seminars in hematology, 2001 - Elsevier