Exposure of HepG2 cells to 1٪(v/v) dimethyl sulfoxide (DMSO), an effective free radical
scavenger, for 24 h resulted in a 2-fold increase in the levels of apolipoprotein (apo) AI …

To induce oxidative stress, HepG2 cells were exposed to a compound known as
gramoxone. This compound undergoes a one-electron reduction to form a stable free radical …

HepG2 cells and Caco-2 cells were treated with various concentrations of select
antioxidants to study some of the molecular pathways underlying antioxidant-related …

The observation that glucocorticoids increase the abundance of apolipoprotein AI led us to a
search for potential underlying mechanism (s). In this report, we show that the synthetic …

Previously, we demonstrated that when two human hepatoma cell lines, Hep3B and HepG2,
were exposed to gemfibrozil, a hypolipidemic drug, a 2-fold induction in apolipoprotein AI …

Treatment with glucocorticoids increases the concentration of plasma high‐density
lipoprotein (HDL), which is inversely correlated to the development of atherosclerosis …

We have previously shown that 17-β-estradiol (E 2) and genistein increase the expression of
apolipoprotein AI (apoA-I), the major protein component of HDL, in Hep G2 cells. To …

Estrogen replacement therapies, such as conjugated equine estrogen (CEE, Premarin®),
reduce the risk of coronary heart disease among postmenopausal women. In the present …

Apolipoprotein (apo) AI, the major protein component of HDL, is synthesized principally in
the small intestine and liver. Recently we observed an increase in plasma apoA-I level in …

Members of the steroid receptor superfamily are known to alter the transcription of
apolipoprotein AI (apo AI), the major apoprotein of high-density lipoprotein (HDL). To assess …