NF-κB family of transcription factors: central regulators of innate and adaptive immune functions
Transcription factors of the Rel/NF-κB family are activated in response to signals that lead to
cell growth, differentiation, and apoptosis, and these proteins are critical elements involved …
cell growth, differentiation, and apoptosis, and these proteins are critical elements involved …
[HTML][HTML] NF-κB Family of Transcription Factors: Central Regulators of Innate and Adaptive Immune Functions
J Caamaño, CA Hunter - Clinical Microbiology Reviews, 2002 - ncbi.nlm.nih.gov
Transcription factors of the Rel/NF-κB family are activated in response to signals that lead to
cell growth, differentiation, and apoptosis, and these proteins are critical elements involved …
cell growth, differentiation, and apoptosis, and these proteins are critical elements involved …
NF-kappaB family of transcription factors: central regulators of innate and adaptive immune functions.
J Caamaño, CA Hunter - Clinical Microbiology Reviews, 2002 - europepmc.org
Transcription factors of the Rel/NF-κB family are activated in response to signals that lead to
cell growth, differentiation, and apoptosis, and these proteins are critical elements involved …
cell growth, differentiation, and apoptosis, and these proteins are critical elements involved …
[引用][C] NF-κB family of Transcription factors: Central regulators of innate and adaptive immune functions
J CAAMANO, CA HUNTER - Clinical microbiology reviews …, 2002 - pascal-francis.inist.fr
NF-κB family of Transcription factors: Central regulators of innate and adaptive immune functions
CNRS Inist Pascal-Francis CNRS Pascal and Francis Bibliographic Databases Simple search …
CNRS Inist Pascal-Francis CNRS Pascal and Francis Bibliographic Databases Simple search …
NF-kB family of transcription factors: Central regulators of innate and adaptive immune functions
J Caamano, CA Hunter - Clinical Microbiology Reviews, 2002 - research.birmingham.ac.uk
Transcription factors of the Rel/NF-kappaB family are activated in response to signals that
lead to cell growth, differentiation, and apoptosis, and these proteins are critical elements …
lead to cell growth, differentiation, and apoptosis, and these proteins are critical elements …
[PDF][PDF] NF-κB Family of Transcription Factors: Central Regulators of Innate and Adaptive Immune Functions
J Caamano, CA Hunter - CLINICAL MICROBIOLOGY REVIEWS, 2002 - Citeseer
In mammals the NF-κB family of transcription factors contains five members: NF-κB1
(p105/p50), NF-κB2 (p100/p52), RelA (p65), RelB, and c-Rel (Fig. 1). NF-κB1 and NF-κB2 …
(p105/p50), NF-κB2 (p100/p52), RelA (p65), RelB, and c-Rel (Fig. 1). NF-κB1 and NF-κB2 …
NF-kappaB family of transcription factors: central regulators of innate and adaptive immune functions
J Caamaño, CA Hunter - Clinical microbiology reviews, 2002 - pubmed.ncbi.nlm.nih.gov
Transcription factors of the Rel/NF-kappaB family are activated in response to signals that
lead to cell growth, differentiation, and apoptosis, and these proteins are critical elements …
lead to cell growth, differentiation, and apoptosis, and these proteins are critical elements …
NF-κB Family of Transcription Factors: Central Regulators of Innate and Adaptive Immune Functions
J Caamaño, CA Hunter - Clinical Microbiology Reviews, 2002 - cir.nii.ac.jp
抄録< jats: sec>< jats: title> SUMMARY</jats: title>< jats: p> Transcription factors of the
Rel/NF-κB family are activated in response to signals that lead to cell growth, differentiation …
Rel/NF-κB family are activated in response to signals that lead to cell growth, differentiation …
[PDF][PDF] B Family of Transcription Factors: κ NF
Citeseer
In mammals the NF-κB family of transcription factors contains five members: NF-κB1
(p105/p50), NF-κB2 (p100/p52), RelA (p65), RelB, and c-Rel (Fig. 1). NF-κB1 and NF-κB2 …
(p105/p50), NF-κB2 (p100/p52), RelA (p65), RelB, and c-Rel (Fig. 1). NF-κB1 and NF-κB2 …
[引用][C] NF-κB family of transcription factors: central regulators of innate and adaptive immune functions
J CAAMANO - Clin. Microbiol. Rev., 2002 - cir.nii.ac.jp