Nap1-mediated actin remodeling is essential for mammalian myoblast fusion

SJ Nowak, PC Nahirney… - Journal of cell …, 2009 - journals.biologists.com
Myoblast fusion is crucial for the formation, growth, maintenance and regeneration of healthy
skeletal muscle. Unfortunately, the molecular machinery, cell behaviors, and membrane and …

[HTML][HTML] Nap1-mediated actin remodeling is essential for mammalian myoblast fusion

SJ Nowak, PC Nahirney, AK Hadjantonakis… - Journal of Cell …, 2009 - ncbi.nlm.nih.gov
Myoblast fusion is crucial for the formation, growth, maintenance and regeneration of healthy
skeletal muscle. Unfortunately, the molecular machinery, cell behaviors, and membrane and …

Nap1-mediated actin remodeling is essential for mammalian myoblast fusion

SJ Nowak, PC Nahirney, AK Hadjantonakis… - Journal of Cell …, 2009 - cir.nii.ac.jp
抄録< jats: p> Myoblast fusion is crucial for the formation, growth, maintenance and
regeneration of healthy skeletal muscle. Unfortunately, the molecular machinery, cell …

Nap1-mediated actin remodeling is essential for mammalian myoblast fusion.

SJ Nowak, PC Nahirney, AK Hadjantonakis… - Journal of Cell …, 2009 - europepmc.org
Myoblast fusion is crucial for the formation, growth, maintenance and regeneration of healthy
skeletal muscle. Unfortunately, the molecular machinery, cell behaviors, and membrane and …

Nap1-mediated actin remodeling is essential for mammalian myoblast fusion

SJ Nowak, PC Nahirney… - Journal of cell …, 2009 - pubmed.ncbi.nlm.nih.gov
Myoblast fusion is crucial for the formation, growth, maintenance and regeneration of healthy
skeletal muscle. Unfortunately, the molecular machinery, cell behaviors, and membrane and …

Nap1-mediated actin remodeling is essential for mammalian myoblast fusion.

SJ Nowak, PC Nahirney, AK Hadjantonakis… - Journal of Cell …, 2009 - europepmc.org
Myoblast fusion is crucial for the formation, growth, maintenance and regeneration of healthy
skeletal muscle. Unfortunately, the molecular machinery, cell behaviors, and membrane and …