Infection of mammalian cells with vesicular stomatitis virus (VSV) results in the inhibition of
cellular translation, while viral translation proceeds efficiently. This host “shutoff” is mediated …

Infection of mammalian cells with vesicular stomatitis virus (VSV) results in the inhibition of
cellular translation while viral translation proceeds efficiently. VSV RNA synthesis occurs …

Vesicular stomatitis virus (VSV) infection causes inhibition of host protein synthesis, in part
by sequestering initiation factors required for mRNA cap recognition. The viral mRNAs share …

Positive-strand and double-strand RNA viruses typically compartmentalize their replication
machinery in infected cells. This is thought to shield viral RNA from detection by innate …

Host protein synthesis is inhibited in cells infected with vesicular stomatitis virus (VSV). It has
been proposed that viral mRNAs are subjected to the same inhibition but are predominantly …

Vesicular stomatitis virus (VSV) modulates protein synthesis in infected cells in a way that
allows the translation of its own 5′-capped mRNA but inhibits the translation of host mRNA …

Cytolytic viruses abrogate host protein synthesis to maximize the translation of their own
mRNAs. In this study, we analyzed the eukaryotic initiation factor (eIF) 4G requirement for …

During vesicular stomatitis virus (VSV) infection, host protein synthesis is inhibited, while
synthesis of viral proteins increases. VSV infection causes inhibition of host transcription and …

In cells that allow replication of vesicular stomatitis virus (VSV), there are two phases of
translation inhibition: an early block of host translation and a later inhibition of viral …

Vesicular stomatitis virus (VSV) mRNAs on polysomes 3 hr after infection are not translated
in vivo as efficiently as mRNAs 2 hr after infection. There are 14−, 12−, 11−, and 8-fold …