Abstract Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common
human enzyme defect, which may present as acute hemolysis, neonatal jaundice, or chronic …

Glucose-6-phosphate dehydrogenase (G6PD) is an enzyme catalyzing the first reaction in
the pentose phosphate pathway to produce NADPH and to fuel glutathione recycling …

Key Clinical Message Aspirin‐related hemolysis in G6PD deficiency could be late‐onset
during long‐term administration. Hemolytic anemia could continue for a relatively long time …

Forty-four patients with Mediterranean-type glucose-6-phosphate dehydrogenase (G-6-PD)
deficiency receiving long-term, low-dose aspirin were monitored over three months for …

Abstract Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common
human enzyme defect and one of the most common genetic disorders worldwide, with an …

Purpose The pathophysiology, diagnosis, and medication-use implications of glucose-6-
phosphate dehydrogenase (G6PD) deficiency, the most common enzyme deficiency in …

Abstract Glucose-6-phospate dehydrogenase (G6PD) deficiency is estimated to affect more
than 400 million people world-wide. This X-linked genetic deficiency puts stress on red …

Glucose-6-phosphate dehydrogenase (G6PD) protects erythrocytes from oxidative stress
and hemolysis; G6PD deficiency is the most prevalent enzymopathy. The United States …

Glucose-6-phosphate dehydrogenase (G6PD) deficiency can present a diagnostic dilemma
owing to the varying degrees of disease severity and the wide range of precipitating factors …

Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common diverse X-
linked human enzymopathy genetic trait. Males are either G6PD-deficient or normal, but …