Counterregulation between thymic stromal lymphopoietin–and IL-23–driven immune axes shapes skin inflammation in mice with epidermal barrier defects

J Li, JM Leyva-Castillo, P Hener, A Eisenmann… - Journal of Allergy and …, 2016 - Elsevier
Background Epidermal barrier dysfunction has been recognized as a critical factor in the
initiation and exacerbation of skin inflammation, particularly in patients with atopic dermatitis …

Counterregulation between thymic stromal lymphopoietin-and IL-23-driven immune axes shapes skin inflammation in mice with epidermal barrier defects

J Li, JM Leyva-Castillo, P Hener… - The Journal of …, 2016 - pubmed.ncbi.nlm.nih.gov
Background Epidermal barrier dysfunction has been recognized as a critical factor in the
initiation and exacerbation of skin inflammation, particularly in patients with atopic dermatitis …

Counterregulation between thymic stromal lymphopoietin-and IL-23-driven immune axes shapes skin inflammation in mice with epidermal barrier defects.

J Li, JM Leyva-Castillo, P Hener… - The Journal of Allergy …, 2016 - europepmc.org
Background Epidermal barrier dysfunction has been recognized as a critical factor in the
initiation and exacerbation of skin inflammation, particularly in patients with atopic dermatitis …

Counterregulation between thymic stromal lymphopoietin–and IL-23–driven immune axes shapes skin inflammation in mice with epidermal barrier defects

J Li, JM Leyva-Castillo, P Hener… - Journal of Allergy …, 2016 - ut3-toulouseinp.hal.science
Background: Epidermal barrier dysfunction has been recognized as a critical factor in the
initiation and exacerbation of skin inflammation, particularly in patients with atopic dermatitis …

Counterregulation between thymic stromal lymphopoietin–and IL-23–driven immune axes shapes skin inflammation in mice with epidermal barrier defects

J Li, JM Leyva-Castillo, P Hener… - Journal of Allergy and …, 2016 - jacionline.org
Background Epidermal barrier dysfunction has been recognized as a critical factor in the
initiation and exacerbation of skin inflammation, particularly in patients with atopic dermatitis …

[PDF][PDF] Counterregulation between thymic stromal lymphopoietin–and IL-23–driven immune axes shapes skin inflammation in mice with epidermal barrier defects

Allergy, 2016 - academia.edu
Background: Epidermal barrier dysfunction has been recognized as a critical factor in the
initiation and exacerbation of skin inflammation, particularly in patients with atopic dermatitis …

Counterregulation between thymic stromal lymphopoietin–and IL-23–driven immune axes shapes skin inflammation in mice with epidermal barrier defects

J Li, JM Leyva-Castillo, P Hener, A Eisenmann… - The Journal of Allergy …, 2016 - infona.pl
Epidermal barrier dysfunction has been recognized as a critical factor in the initiation and
exacerbation of skin inflammation, particularly in patients with atopic dermatitis (AD) and AD …

[引用][C] Counterregulation between thymic stromal lymphopoietin–and IL-23–driven immune axes shapes skin inflammation in mice with epidermal barrier defects

J Li, JM Leyva-Castillo, P Hener, A Eisenmann… - Journal of Allergy and …, 2016 - cir.nii.ac.jp
Counterregulation between thymic stromal lymphopoietin– and IL-23–driven immune axes
shapes skin inflammation in mice with epidermal barrier defects | CiNii Research CiNii 国立 …

Counterregulation between thymic stromal lymphopoietin–and IL-23–driven immune axes shapes skin inflammation in mice with epidermal barrier defects

J Li, JM Leyva-Castillo, P Hener… - Journal of Allergy and …, 2016 - hal.science
Background: Epidermal barrier dysfunction has been recognized as a critical factor in the
initiation and exacerbation of skin inflammation, particularly in patients with atopic dermatitis …

[PDF][PDF] Counterregulation between thymic stromal lymphopoietin–and IL-23–driven immune axes shapes skin inflammation in mice with epidermal barrier defects

Allergy, 2016 - academia.edu
Background: Epidermal barrier dysfunction has been recognized as a critical factor in the
initiation and exacerbation of skin inflammation, particularly in patients with atopic dermatitis …