[HTML][HTML] Monoallelic intragenic POU3F2 variants lead to neurodevelopmental delay and hyperphagic obesity, confirming the gene's candidacy in 6q16. 1 deletions

R Schönauer, W Jin, C Findeisen, I Valenzuela… - The American Journal of …, 2023 - cell.com
While common obesity accounts for an increasing global health burden, its monogenic forms
have taught us underlying mechanisms via more than 20 single-gene disorders. Among …

Monoallelic intragenic POU3F2 variants lead to neurodevelopmental delay and hyperphagic obesity, confirming the gene's candidacy in 6q16. 1 deletions.

R Schönauer, W Jin, C Findeisen… - American Journal of …, 2023 - europepmc.org
While common obesity accounts for an increasing global health burden, its monogenic forms
have taught us underlying mechanisms via more than 20 single-gene disorders. Among …

Monoallelic intragenic POU3F2 variants lead to neurodevelopmental delay and hyperphagic obesity, confirming the gene's candidacy in 6q16.1 deletions

R Schonauer, W Jin, C Findeisen… - American Journal of …, 2023 - eprints.ncl.ac.uk
Abstract© 2023 American Society of Human GeneticsWhile common obesity accounts for an
increasing global health burden, its monogenic forms have taught us underlying …

Monoallelic intragenic POU3F2 variants lead to neurodevelopmental delay and hyperphagic obesity, confirming the gene's candidacy in 6q16. 1 deletions

R Schönauer, W Jin, C Findeisen… - … Journal of Human …, 2023 - research-collection.ethz.ch
While common obesity accounts for an increasing global health burden, its monogenic forms
have taught us underlying mechanisms via more than 20 single-gene disorders. Among …

[HTML][HTML] Monoallelic intragenic POU3F2 variants lead to neurodevelopmental delay and hyperphagic obesity, confirming the gene's candidacy in 6q16. 1 deletions

R Schönauer, W Jin, C Findeisen, I Valenzuela… - The American Journal of …, 2023 - Elsevier
While common obesity accounts for an increasing global health burden, its monogenic forms
have taught us underlying mechanisms via more than 20 single-gene disorders. Among …

Monoallelic intragenic POU3F2 variants lead to neurodevelopmental delay and hyperphagic obesity, confirming the gene's candidacy in 6q16. 1 deletions

R Schönauer, W Jin, C Findeisen… - … journal of human …, 2023 - pubmed.ncbi.nlm.nih.gov
While common obesity accounts for an increasing global health burden, its monogenic forms
have taught us underlying mechanisms via more than 20 single-gene disorders. Among …

[HTML][HTML] Monoallelic intragenic POU3F2 variants lead to neurodevelopmental delay and hyperphagic obesity, confirming the gene's candidacy in 6q16. 1 deletions

R Schönauer, W Jin, C Findeisen… - American Journal of …, 2023 - ncbi.nlm.nih.gov
While common obesity accounts for an increasing global health burden, its monogenic forms
have taught us underlying mechanisms via more than 20 single-gene disorders. Among …

Monoallelic intragenic POU3F2 variants lead to neurodevelopmental delay and hyperphagic obesity, confirming the gene's candidacy in 6q16. 1 deletions

R Schönauer, W Jin, C Findeisen… - … Journal of Human …, 2023 - researchportal.helsinki.fi
Monoallelic intragenic POU3F2 variants lead to neurodevelopmental delay and hyperphagic
obesity, confirming the gene's candidacy in 6q16.1 deletions — University of Helsinki Skip to …

Monoallelic intragenic POU3F2 variants lead to neurodevelopmental delay and hyperphagic obesity, confirming the gene's candidacy in 6q16. 1 deletions.

R Schönauer, W Jin, C Findeisen… - American Journal of …, 2023 - hal.science
While common obesity accounts for an increasing global health burden, its monogenic forms
have taught us underlying mechanisms via more than 20 single-gene disorders. Among …

Monoallelic intragenic POU3F2 variants lead to neurodevelopmental delay and hyperphagic obesity, confirming the gene's candidacy in 6q16. 1 deletions.

R Schönauer, W Jin, C Findeisen, I Valenzuela… - 2023 - mediatum.ub.tum.de
While common obesity accounts for an increasing global health burden, its monogenic forms
have taught us underlying mechanisms via more than 20 single-gene disorders. Among …