Biologics and their delivery systems: Trends in myocardial infarction
Cardiovascular disease is the leading cause of death around the world, in which myocardial
infarction (MI) is a precipitating event. However, current therapies do not adequately address …
infarction (MI) is a precipitating event. However, current therapies do not adequately address …
The NLRP3 inflammasome: multiple activation pathways and its role in primary cells during ventricular remodeling
Z Yan, Z Qi, X Yang, N Ji, Y Wang, Q Shi… - Journal of Cellular …, 2021 - Wiley Online Library
Inflammasomes are a group of multiprotein signaling complexes located in the cytoplasm.
Several inflammasomes have been identified, including NLRP1, NLRP2, NLRP3, AIM2, and …
Several inflammasomes have been identified, including NLRP1, NLRP2, NLRP3, AIM2, and …
Macrophages promote endothelial-to-mesenchymal transition via MT1-MMP/TGFβ1 after myocardial infarction
L Alonso-Herranz, Á Sahún-Español, A Paredes… - Elife, 2020 - elifesciences.org
Macrophages (Mφs) produce factors that participate in cardiac repair and remodeling after
myocardial infarction (MI); however, how these factors crosstalk with other cell types …
myocardial infarction (MI); however, how these factors crosstalk with other cell types …
[HTML][HTML] The roles of noncardiomyocytes in cardiac remodeling
D Yang, HQ Liu, FY Liu, N Tang, Z Guo… - … journal of biological …, 2020 - ncbi.nlm.nih.gov
Cardiac remodeling is a common characteristic of almost all forms of heart disease,
including cardiac infarction, valvular diseases, hypertension, arrhythmia, dilated …
including cardiac infarction, valvular diseases, hypertension, arrhythmia, dilated …
[HTML][HTML] TIMP3 induces gene expression partly through PI3K and their association with vascularization and heart rate
ZM Xia, MY Song, YL Chen, G Cui… - Frontiers in Cardiovascular …, 2023 - frontiersin.org
Background: Tissue inhibitor of metalloproteinase 3 (TIMP3) was recently demonstrated
capable to regulate some gene expression in a myocardial infarction model. Here we aim to …
capable to regulate some gene expression in a myocardial infarction model. Here we aim to …
Investigation of the pharmacological effect and mechanism of mountain-cultivated ginseng and garden ginseng in cardiovascular diseases based on network …
T Yu, YX Zhang, XJ Liu, DQ Chen, DD Wang… - Frontiers in …, 2022 - frontiersin.org
Ginseng (Panax ginseng CA Mey) is a kind of perennial herb of the Panax genus in the
Araliaceae family. The secondary metabolites of mountain-cultivated ginseng (MCG) and …
Araliaceae family. The secondary metabolites of mountain-cultivated ginseng (MCG) and …
Variation in VEGFA and risk of cardiovascular disease in the UK Biobank
H Chen, X Lv, J Yang, Z Chen, W Qiao… - Frontiers in …, 2023 - frontiersin.org
Background: Cardiovascular disease (CVD) is an escalating global health crisis,
contributing significantly to worldwide mortality and morbidity. Dyslipidemia stands as a …
contributing significantly to worldwide mortality and morbidity. Dyslipidemia stands as a …
[HTML][HTML] Characterization of encapsulated porcine cardiosphere-derived cells embedded in 3D alginate matrices
K Ziani, A Espona-Noguera, V Crisóstomo… - International journal of …, 2021 - Elsevier
Myocardial infarction is caused by an interruption of coronary blood flow, leading to one of
the main death causes worldwide. Current therapeutic approaches are palliative and not …
the main death causes worldwide. Current therapeutic approaches are palliative and not …
[HTML][HTML] 1-磷酸鞘氨醇1 型受体促进小鼠颈动脉损伤后血管内膜增生
任宏强， 刘益均， 罗贵全， 赵利 - 2021 - aammt.tmmu.edu.cn
目的探究1-磷酸鞘氨醇1 型受体(sphingosine-1-phosphate type 1 receptor, S1PR1)
对小鼠颈动脉损伤后血管内膜增生的作用及其机制. 方法40 只C57BL/6 小鼠按随机数字表法分 …
对小鼠颈动脉损伤后血管内膜增生的作用及其机制. 方法40 只C57BL/6 小鼠按随机数字表法分 …