Objectives: During the advancement of atherosclerosis, plaque cellularity is governed by the
influx of monocyte-derived macrophages and their turnover via apoptotic and nonapoptotic …

Background Human coagulation factor (F) XI deficiency, a defect of the contact activation
system, protects against venous thrombosis, stroke, and heart attack, whereas FXII, plasma …

Objective: Although the molecular components of circadian rhythms oscillate in discrete
cellular components of the vasculature and many aspects of vascular function display …

Background: Insufficient or disrupted sleep increases the risk of cardiovascular disease,
including atherosclerosis. However, we know little about the molecular mechanisms by …

CXC chemokine ligand 12 (CXCL12) is a member of the CXC chemokine family and mainly
acts on cell chemotaxis. CXCL12 also elicits a proatherogenic role, but the molecular …

It has been shown that SGLT2 suppresses atherosclerosis (AS). Recent studies indicate that
autophagy widely participates in atherogenesis. This study aimed to assess the effect of …

Objective: LDL (low-density lipoprotein) transcytosis across the endothelium is performed by
the SR-BI (scavenger receptor class B type 1) receptor and contributes to atherosclerosis …

Objective: ODC (ornithine decarboxylase)-dependent putrescine synthesis promotes the
successive clearance of apoptotic cells (ACs) by macrophages, contributing to inflammation …

Objective—Increasing evidence shows that resveratrol has antiatherogenic effects, but its
underlying mechanisms are unknown. Thus, we evaluated the molecular mechanisms …

Aging is a strong risk factor for atherosclerosis and induces accumulation of memory CD8+ T
cells in mice and humans. Biological changes that occur with aging lead to enhanced …