[HTML][HTML] SARS-CoV-2-specific immune response and the pathogenesis of COVID-19
E Gusev, A Sarapultsev, L Solomatina… - International journal of …, 2022 - mdpi.com
The review aims to consolidate research findings on the molecular mechanisms and
virulence and pathogenicity characteristics of coronavirus disease (COVID-19) causative …
virulence and pathogenicity characteristics of coronavirus disease (COVID-19) causative …
[HTML][HTML] The roles of Eph receptors, neuropilin-1, P2X7, and CD147 in COVID-19-associated neurodegenerative diseases: inflammasome and JaK inhibitors as …
H Zalpoor, A Akbari, A Samei… - Cellular & Molecular …, 2022 - Springer
The novel coronavirus disease 2019 (COVID-19) pandemic has spread worldwide, and
finding a safe therapeutic strategy and effective vaccine is critical to overcoming severe …
finding a safe therapeutic strategy and effective vaccine is critical to overcoming severe …
A scoping review of the pathophysiology of COVID-19
COVID-19 is a highly heterogeneous and complex medical disorder; indeed, severe COVID-
19 is probably amongst the most complex of medical conditions known to medical science …
19 is probably amongst the most complex of medical conditions known to medical science …
[HTML][HTML] T cell metabolism in infection
JA Wik, BS Skålhegg - Frontiers in immunology, 2022 - frontiersin.org
T lymphocytes (T cells) are divided into two functionally different subgroups the CD4+ T
helper cells (Th) and the CD8+ cytotoxic T lymphocytes (CTL). Adequate CD4 and CD8 T …
helper cells (Th) and the CD8+ cytotoxic T lymphocytes (CTL). Adequate CD4 and CD8 T …
Neuropilin-1 mediates SARS-CoV-2 infection of astrocytes in brain organoids, inducing inflammation leading to dysfunction and death of neurons
ABSTRACT Coronavirus disease 2019 (COVID-19) is frequently associated with
neurological deficits, but how severe acute respiratory syndrome coronavirus 2 (SARS-CoV …
neurological deficits, but how severe acute respiratory syndrome coronavirus 2 (SARS-CoV …
[HTML][HTML] Structural and non-structural proteins in SARS-CoV-2: potential aspects to COVID-19 treatment or prevention of progression of related diseases
S Kakavandi, I Zare, M VaezJalali, M Dadashi… - Cell Communication and …, 2023 - Springer
Abstract Coronavirus disease 2019 (COVID-19) is caused by a new member of the
Coronaviridae family known as severe acute respiratory syndrome coronavirus 2 (SARS …
Coronaviridae family known as severe acute respiratory syndrome coronavirus 2 (SARS …
Covid-19-induced dysautonomia: a menace of sympathetic storm
Among the plethora of debilitating neurological disorders of COVID-19 syndrome in
survivors, the scope of SARS-CoV-2-induced dysautonomia (DNS) is yet to be understood …
survivors, the scope of SARS-CoV-2-induced dysautonomia (DNS) is yet to be understood …
[HTML][HTML] SARS-CoV-2 interacts with platelets and megakaryocytes via ACE2-independent mechanism
S Shen, J Zhang, Y Fang, S Lu, J Wu, X Zheng… - Journal of hematology & …, 2021 - Springer
Evidence suggests that platelets may directly interact with SARS-CoV-2, raising the concern
whether ACE2 receptor plays a role in this interaction. The current study showed that SARS …
whether ACE2 receptor plays a role in this interaction. The current study showed that SARS …
[HTML][HTML] Endothelial contribution to COVID-19: an update on mechanisms and therapeutic implications
The global propagation of SARS-CoV-2 leads to an unprecedented public health
emergency. Despite that the lungs are the primary organ targeted by COVID-19, systemic …
emergency. Despite that the lungs are the primary organ targeted by COVID-19, systemic …
[HTML][HTML] SARS-CoV-2 cell entry beyond the ACE2 receptor
SD Alipoor, M Mirsaeidi - Molecular biology reports, 2022 - Springer
Background Angiotensin-converting enzyme 2 (ACE2) is known as the major viral entry site
for SARS-CoV-2. However, viral tissue tropism and high rate of infectivity do not directly …
for SARS-CoV-2. However, viral tissue tropism and high rate of infectivity do not directly …