Ferroptosis is an iron-dependent regulated cell death driven by excessive lipid peroxidation.
Inflammation is one common and effective physiological event that protects against various …

Abstract Ferroptosis suppressor protein 1 (FSP1) is one of the main regulatory molecules of
ferroptosis. FSP1 functions through the FSP1-coenzyme Q10 (CoQ10)-NAD (P) H axis and …

Ferroptosis, a unique modality of cell death with mechanistic and morphological differences
from other cell death modes, plays a pivotal role in regulating tumorigenesis and offers a …

Neutrophil extracellular traps (NETs) play an important role in abdominal aortic aneurysm
(AAA) formation; however, the underlying molecular mechanisms remain unclear …

Abstract Background E1A-associated 300-kDa protein (P300), an endogenous histone
acetyltransferase, contributes to modifications of the chromatin landscape of genes involved …

Ferroptosis is a form of programmed cell death characterized by elevated intracellular
ferrous ion levels and increased lipid peroxidation. Since its discovery and characterization …

Cardiovascular disease (CVD) is the leading cause of death in the world, with a high
incidence and a youth-oriented tendency. RNA modification is ubiquitous and indispensable …

Rationale Myocardial ischemia/reperfusion (I/R) injury is characterized by cell death via
various cellular mechanisms upon reperfusion. As a new type of cell death, ferroptosis …

Prevention of neointima formation is the key to improving long‐term outcomes after stenting
or coronary artery bypass grafting. RNA N6‐methyladenosine (m6A) methylation has been …

Iron functions as an essential micronutrient and participates in normal physiological and
biochemical processes in the cardiovascular system. Ferroptosis is a novel type of iron …