Alzheimer's disease (AD) is a debilitating disorder that manifests with amyloid beta plaque
deposition, neurofibrillary tangles, neuronal loss, and severe cognitive impairment. Although …

The molecular biological mechanisms of Alzheimer's disease (AD) involve disease-
associated crosstalk through many genes and include a loss of normal as well as a gain of …

Deposition of amyloid‐β (Aβ) as senile plaques is one of the pathological hallmarks in the
brains of Alzheimer's disease (AD) patients. In addition, glial activation has been found in …

The immediate early gene Arc is a master regulator of synaptic function and a critical
determinant of memory consolidation. Here, we show that Arc interacts with dynamic …

Alzheimer's disease (AD) is the most common type of dementia, with increasing prevalence
and no disease‐modifying treatment available yet. There is increasing evidence—from …

Hypoxia, that is, an inadequate oxygen supply, is linked to neurodegeneration and patients
with cardiovascular disease are prone to Alzheimer's disease (AD). 2‐Oxoglutarate and …

Alzheimer's disease (AD) is the most common neurodegenerative disorder. Extracellular
vesicles (EVs), carriers of nucleic acids, lipids, and proteins, are known to play significant …

Age-related pathologies like Alzheimers disease (AD) imply cellular responses directed
towards repairing DNA damage. Postmitotic neurons show progressive accumulation of …

The amyloid precursor protein (APP) has been extensively studied as the precursor of the β-
amyloid (Aβ) peptide, the major component of the senile plaques found in the brain of …

Hippocampal neuronal activity generates dendritic and somatic Ca2+ signals, which,
depending on stimulus intensity, rapidly propagate to the nucleus and induce the expression …