Confronting COVID-19-associated cough and the post-COVID syndrome: role of viral neurotropism, neuroinflammation, and neuroimmune responses
Cough is one of the most common presenting symptoms of COVID-19, along with fever and
loss of taste and smell. Cough can persist for weeks or months after SARS-CoV-2 infection …
loss of taste and smell. Cough can persist for weeks or months after SARS-CoV-2 infection …
[HTML][HTML] Neurological complications and infection mechanism of SARS-COV-2
D Wan, T Du, W Hong, L Chen, H Que, S Lu… - Signal Transduction and …, 2021 - nature.com
Abstract Currently, SARS-CoV-2 has caused a global pandemic and threatened many lives.
Although SARS-CoV-2 mainly causes respiratory diseases, growing data indicate that SARS …
Although SARS-CoV-2 mainly causes respiratory diseases, growing data indicate that SARS …
[HTML][HTML] SARS-CoV-2 infects cells after viral entry via clathrin-mediated endocytosis
Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is the causative agent of
COVID-19, so understanding its biology and infection mechanisms is critical to facing this …
COVID-19, so understanding its biology and infection mechanisms is critical to facing this …
[HTML][HTML] Neuroinflammation and COVID-19
A Vanderheiden, RS Klein - Current opinion in neurobiology, 2022 - Elsevier
Abstract Coronavirus disease 2019 (COVID-19) has caused a historic pandemic of
respiratory disease. COVID-19 also causes acute and post-acute neurological symptoms …
respiratory disease. COVID-19 also causes acute and post-acute neurological symptoms …
[HTML][HTML] The roles of Eph receptors, neuropilin-1, P2X7, and CD147 in COVID-19-associated neurodegenerative diseases: inflammasome and JaK inhibitors as …
H Zalpoor, A Akbari, A Samei… - Cellular & Molecular …, 2022 - Springer
The novel coronavirus disease 2019 (COVID-19) pandemic has spread worldwide, and
finding a safe therapeutic strategy and effective vaccine is critical to overcoming severe …
finding a safe therapeutic strategy and effective vaccine is critical to overcoming severe …
[HTML][HTML] SARS-CoV-2 and the host cell: a tale of interactions
M Pizzato, C Baraldi, G Boscato Sopetto… - Frontiers in …, 2022 - frontiersin.org
The ability of a virus to spread between individuals, its replication capacity and the clinical
course of the infection are macroscopic consequences of a multifaceted molecular …
course of the infection are macroscopic consequences of a multifaceted molecular …
[HTML][HTML] The COVID-19 pandemic and Alzheimer's disease: mutual risks and mechanisms
Abstract Coronavirus disease 2019 (COVID-19), which is caused by severe acute
respiratory syndrome coronavirus 2 (SARS-CoV-2), is a life-threatening disease, especially …
respiratory syndrome coronavirus 2 (SARS-CoV-2), is a life-threatening disease, especially …
[HTML][HTML] Differential effects of SARS-CoV-2 variants on central nervous system cells and blood–brain barrier functions
A Proust, CJ Queval, R Harvey, L Adams… - Journal of …, 2023 - Springer
Background Although mainly causing a respiratory syndrome, numerous neurological
symptoms have been identified following of SARS-CoV-2 infection. However, how the virus …
symptoms have been identified following of SARS-CoV-2 infection. However, how the virus …
Covid-19-induced dysautonomia: a menace of sympathetic storm
Among the plethora of debilitating neurological disorders of COVID-19 syndrome in
survivors, the scope of SARS-CoV-2-induced dysautonomia (DNS) is yet to be understood …
survivors, the scope of SARS-CoV-2-induced dysautonomia (DNS) is yet to be understood …
[HTML][HTML] Not only ACE2—the quest for additional host cell mediators of SARS-CoV-2 infection: Neuropilin-1 (NRP1) as a novel SARS-CoV-2 host cell entry mediator …
I Kyrou, HS Randeva, DA Spandidos… - Signal transduction and …, 2021 - nature.com
Two recently published studies published in Science by Daly et al. 1 and Cantuti-Castelvetri
et al. 2 identified neuropilin-1 (NRP1) as an additional cellular mediator which may facilitate …
et al. 2 identified neuropilin-1 (NRP1) as an additional cellular mediator which may facilitate …