Yellon, Derek M., and James M. Downey. Preconditioning the Myocardium: From Cellular
Physiology to Clinical Cardiology. Physiol Rev 83: 1113-1151, 2003; 10.1152/physrev …

The biochemical mechanisms behind skeletal muscle soreness and damage with muscular
overuse have remained unclear. Recently, however, a growing amount of evidence …

Background—Serum uric acid (UA) could be a valid prognostic marker and useful for
metabolic, hemodynamic, and functional (MFH) staging in chronic heart failure (CHF) …

Work during the past several years has produced new insights into many aspects of the cell
biology of ischemic injury to the tubulointerstitium. The major processes are conveniently …

It has been suggested that the sudden presence of oxygen during reperfusion after a period
of ischemia may be toxic for the myocardial cell. The oxygen molecule is capable of …

Direct cytotoxic effects associated with hemoglobin (Hb) or myoglobin (Mb) have been
ascribed to redox reactions (involving either one-or two-electron steps) between the heme …

Myocardial ischemia causes heart injury that is characterized by an increase in circulating
tumor necrosis factor (TNF), the local production of superoxide anions, the loss of coronary …

Study objective Hyperuricemia occurs frequently inpatients with myeloproliferative and
lymphoproliferative disorders andin patients with congenital heart disease associated with …

Objective. This article reviews the biochemistry and function of xanthine dehydrogenase
(XDH) and xanthine oxidase (XO) as well as their role in hypoxia-reoxygenation injury …

In recent years there has been considerable interest concerning the role of oxygen radicals
in myocardial ischemia and reperfusion injury. The sequential univalent reduction of oxygen …