[HTML][HTML] Anti-apoptosis and cell survival: a review

L Portt, G Norman, C Clapp, M Greenwood… - … et Biophysica Acta (BBA …, 2011 - Elsevier
Type I programmed cell death (PCD) or apoptosis is critical for cellular self-destruction for a
variety of processes such as development or the prevention of oncogenic transformation …

[HTML][HTML] Mitochondrial channels: ion fluxes and more

I Szabo, M Zoratti - Physiological reviews, 2014 - journals.physiology.org
The field of mitochondrial ion channels has recently seen substantial progress, including the
molecular identification of some of the channels. An integrative approach using genetics …

KATP Channels in the Cardiovascular System

MN Foster, WA Coetzee - Physiological reviews, 2016 - journals.physiology.org
KATP channels are integral to the functions of many cells and tissues. The use of
electrophysiological methods has allowed for a detailed characterization of KATP channels …

The role of heat shock proteins and co-chaperones in heart failure

MJ Ranek, MJ Stachowski… - … Transactions of the …, 2018 - royalsocietypublishing.org
The ongoing contractile and metabolic demands of the heart require a tight control over
protein quality control, including the maintenance of protein folding, turnover and synthesis …

Resolvin E1 protects the rat heart against reperfusion injury

KT Keyes, Y Ye, Y Lin, C Zhang… - American Journal …, 2010 - journals.physiology.org
The purpose of the present study was to assess whether resolvin E1 (RvE1), an anti-
inflammatory mediator derived from eicosapentaenoic acid, would limit myocardial infarct …

Resveratrol increase myocardial Nrf2 expression in type 2 diabetic rats and alleviate myocardial ischemia/reperfusion injury (MIRI)

G Xu, X Zhao, J Fu, X Wang - Annals of palliative medicine, 2019 - apm.amegroups.org
Background: Oxidative stress is one of the main factors that increases reperfusion injury in
the diabetic heart. Resveratrol could decrease oxidation and promote antioxidant factor …

Upregulation of Hsp27 via further inhibition of histone H2A ubiquitination confers protection against myocardial ischemia/reperfusion injury by promoting glycolysis …

P Shi, J Wu, M Li, Y Cao, J Wu, P Ren, K Liu… - Cell Death …, 2023 - nature.com
Research suggests that ischemic glycolysis improves myocardial tolerance to anoxia and
low-flow ischemia. The rate of glycolysis during ischemia reflects the severity of the injury …

Activation of PKA, p38 MAPK and ERK1/2 by gonadotropins in cumulus cells is critical for induction of EGF-like factor and TACE/ADAM17 gene expression during in …

Y Yamashita, M Hishinuma, M Shimada - Journal of ovarian research, 2009 - Springer
Objectives During ovulation, it has been shown that LH stimulus induces the expression of
numerous genes via PKA, p38 MAPK, PI3K and ERK1/2 in cumulus cells and granulosa …

Hypothermic organ preservation by static storage methods: Current status and a view to the future

JF McAnulty - Cryobiology, 2010 - Elsevier
The donor organ shortage is the largest problem in transplantation today and is one where
organ preservation technology has an important role to play. Static storage of solid organs …

Inhibition of p38 MAPK activation protects cardiac mitochondria from ischemia/reperfusion injury

S Kumphune, S Surinkaew, SC Chattipakorn… - Pharmaceutical …, 2015 - Taylor & Francis
Context: Cardiac cell death and fatal arrhythmias during myocardial ischemia/reperfusion
(I/R) can be reduced by p38 MAPK inhibition. However, the effects of p38 MAPK inhibition …