Background The inflammatory response occurring in acute myocardial infarction (AMI) has
been proposed as a potential pharmacological target. Sodium-glucose co-transporter 2 …

Vagus nerve stimulation (VNS) has been found to exert anti‐inflammatory effects in different
clinical settings and has been associated with improvement of clinical outcomes. However …

Background SGTL2-inhibitors are a cornerstone in the treatment of heart failure, but data on
patients with acute myocardial infarction (AMI) is limited. The EMMY trial was the first to …

Myocardial infarction (MI) is irreversible damage to the myocardial tissue caused by
prolonged ischemia/hypoxia, subsequently leading to loss of contractile function and …

Objective Inflammatory cytokines modulate atherogenesis and plaque rupture to involve in
ST‐segment elevation myocardial infarction (STEMI) progression. The present study …

Objectives Inflammatory pathways play a significant role in atherosclerosis that leads to
acute coronary syndrome (ACS). The initial stages of atherosclerosis are often …

Mitochondrial dysfunction and the activation of multiple programmed cell death (PCD) have
been shown to aggravate the severity and mortality associated with the progression of …

Purpose Elevated levels of the inflammatory marker interleukin-6 (IL-6) and cardiac injury
marker N-terminal pro-B-type natriuretic peptide (NT-proBNP) have been observed in …

In recent decades, there has been a progressive understanding of the complex interplay
between cytokines and chemokines released after an acute myocardial infarction (AMI) and …

The gap in excess mortality between subjects with and without diabetes has not decreased
over time. The aim of this study was to investigate the determinants of mortality after acute …